Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug.
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| Title: | Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug. |
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| Authors: | Barak B; McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA. boazba@tauex.tau.ac.il.; The School of Psychological Sciences, Faculty of Social Sciences, Tel Aviv University, Tel Aviv, Israel. boazba@tauex.tau.ac.il.; The Sagol School of Neuroscience, Tel Aviv University, Tel Aviv, Israel. boazba@tauex.tau.ac.il., Zhang Z; F.M. Kirby Neurobiology Center, Boston Children's Hospital and Department of Neurology, Harvard Medical School, Boston, MA, USA., Liu Y; F.M. Kirby Neurobiology Center, Boston Children's Hospital and Department of Neurology, Harvard Medical School, Boston, MA, USA., Nir A; The Sagol School of Neuroscience, Tel Aviv University, Tel Aviv, Israel., Trangle SS; The School of Psychological Sciences, Faculty of Social Sciences, Tel Aviv University, Tel Aviv, Israel., Ennis M; McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA., Levandowski KM; Stanley Center for Psychiatric Research, Broad Institute of MIT and Harvard, Cambridge, MA, USA., Wang D; McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA., Quast K; McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA., Boulting GL; Department of Neurobiology, Harvard Medical School, Boston, MA, USA., Li Y; F.M. Kirby Neurobiology Center, Boston Children's Hospital and Department of Neurology, Harvard Medical School, Boston, MA, USA., Bayarsaihan D; Department of Reconstructive Sciences, University of Connecticut, Farmington, CT, USA., He Z; F.M. Kirby Neurobiology Center, Boston Children's Hospital and Department of Neurology, Harvard Medical School, Boston, MA, USA. zhigang.he@childrens.harvard.edu., Feng G; McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA. fengg@mit.edu.; Stanley Center for Psychiatric Research, Broad Institute of MIT and Harvard, Cambridge, MA, USA. fengg@mit.edu. |
| Source: | Nature neuroscience [Nat Neurosci] 2019 May; Vol. 22 (5), pp. 700-708. Date of Electronic Publication: 2019 Apr 22. |
| Publication Type: | Journal Article; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: Nature Publishing Group Country of Publication: United States NLM ID: 9809671 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1546-1726 (Electronic) Linking ISSN: 10976256 NLM ISO Abbreviation: Nat Neurosci Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
| FullText | Text: Availability: 0 |
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| Header | DbId: mdl DbLabel: MEDLINE Ultimate An: 31011227 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AU" term="%22Barak+B%22">Barak B</searchLink>; McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA. boazba@tauex.tau.ac.il.; The School of Psychological Sciences, Faculty of Social Sciences, Tel Aviv University, Tel Aviv, Israel. boazba@tauex.tau.ac.il.; The Sagol School of Neuroscience, Tel Aviv University, Tel Aviv, Israel. boazba@tauex.tau.ac.il.<br /><searchLink fieldCode="AU" term="%22Zhang+Z%22">Zhang Z</searchLink>; F.M. Kirby Neurobiology Center, Boston Children's Hospital and Department of Neurology, Harvard Medical School, Boston, MA, USA.<br /><searchLink fieldCode="AU" term="%22Liu+Y%22">Liu Y</searchLink>; F.M. Kirby Neurobiology Center, Boston Children's Hospital and Department of Neurology, Harvard Medical School, Boston, MA, USA.<br /><searchLink fieldCode="AU" term="%22Nir+A%22">Nir A</searchLink>; The Sagol School of Neuroscience, Tel Aviv University, Tel Aviv, Israel.<br /><searchLink fieldCode="AU" term="%22Trangle+SS%22">Trangle SS</searchLink>; The School of Psychological Sciences, Faculty of Social Sciences, Tel Aviv University, Tel Aviv, Israel.<br /><searchLink fieldCode="AU" term="%22Ennis+M%22">Ennis M</searchLink>; McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA.<br /><searchLink fieldCode="AU" term="%22Levandowski+KM%22">Levandowski KM</searchLink>; Stanley Center for Psychiatric Research, Broad Institute of MIT and Harvard, Cambridge, MA, USA.<br /><searchLink fieldCode="AU" term="%22Wang+D%22">Wang D</searchLink>; McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA.<br /><searchLink fieldCode="AU" term="%22Quast+K%22">Quast K</searchLink>; McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA.<br /><searchLink fieldCode="AU" term="%22Boulting+GL%22">Boulting GL</searchLink>; Department of Neurobiology, Harvard Medical School, Boston, MA, USA.<br /><searchLink fieldCode="AU" term="%22Li+Y%22">Li Y</searchLink>; F.M. Kirby Neurobiology Center, Boston Children's Hospital and Department of Neurology, Harvard Medical School, Boston, MA, USA.<br /><searchLink fieldCode="AU" term="%22Bayarsaihan+D%22">Bayarsaihan D</searchLink>; Department of Reconstructive Sciences, University of Connecticut, Farmington, CT, USA.<br /><searchLink fieldCode="AU" term="%22He+Z%22">He Z</searchLink>; F.M. Kirby Neurobiology Center, Boston Children's Hospital and Department of Neurology, Harvard Medical School, Boston, MA, USA. zhigang.he@childrens.harvard.edu.<br /><searchLink fieldCode="AU" term="%22Feng+G%22">Feng G</searchLink>; McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA. fengg@mit.edu.; Stanley Center for Psychiatric Research, Broad Institute of MIT and Harvard, Cambridge, MA, USA. fengg@mit.edu. – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%229809671%22">Nature neuroscience</searchLink> [Nat Neurosci] 2019 May; Vol. 22 (5), pp. 700-708. <i>Date of Electronic Publication: </i>2019 Apr 22. – Name: TypePub Label: Publication Type Group: TypPub Data: Journal Article; Research Support, Non-U.S. Gov't – Name: TitleSource Label: Journal Info Group: Src Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Nature+Publishing+Group%22">Nature Publishing Group </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>9809671 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1546-1726 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2210976256%22">10976256 </searchLink><i>NLM ISO Abbreviation: </i>Nat Neurosci <i>Subsets: </i>MEDLINE |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=31011227 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.1038/s41593-019-0380-9 Languages: – Code: eng Text: English PhysicalDescription: Pagination: StartPage: 700 Titles: – TitleFull: Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Barak B – PersonEntity: Name: NameFull: Zhang Z – PersonEntity: Name: NameFull: Liu Y – PersonEntity: Name: NameFull: Nir A – PersonEntity: Name: NameFull: Trangle SS – PersonEntity: Name: NameFull: Ennis M – PersonEntity: Name: NameFull: Levandowski KM – PersonEntity: Name: NameFull: Wang D – PersonEntity: Name: NameFull: Quast K – PersonEntity: Name: NameFull: Boulting GL – PersonEntity: Name: NameFull: Li Y – PersonEntity: Name: NameFull: Bayarsaihan D – PersonEntity: Name: NameFull: He Z – PersonEntity: Name: NameFull: Feng G IsPartOfRelationships: – BibEntity: Dates: – D: 01 M: 05 Text: 2019 May Type: published Y: 2019 Identifiers: – Type: issn-electronic Value: 1546-1726 Numbering: – Type: volume Value: 22 – Type: issue Value: 5 Titles: – TitleFull: Nature neuroscience Type: main |
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