Semaphorin 3E/PlexinD1 signaling is required for cardiac ventricular compaction.

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Title: Semaphorin 3E/PlexinD1 signaling is required for cardiac ventricular compaction.
Authors: Sandireddy R; Program in Cardiovascular and Metabolic Disorders, Duke-NUS Medical School Singapore, Singapore., Cibi DM; Program in Cardiovascular and Metabolic Disorders, Duke-NUS Medical School Singapore, Singapore., Gupta P; Program in Cardiovascular and Metabolic Disorders, Duke-NUS Medical School Singapore, Singapore., Singh A; Program in Cardiovascular and Metabolic Disorders, Duke-NUS Medical School Singapore, Singapore., Tee N; National Heart Research Institute Singapore, National Heart Center Singapore, Singapore., Uemura A; Department of Retinal Vascular Biology, Nagoya City University Graduate School of Medical Sciences, Mizuho-ku, Nagoya, Japan., Epstein JA; Penn Cardiovascular Institute, Department of Medicine, Department of Cell and Developmental Biology, and Institute for Regenerative Medicine, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, USA., Singh MK; Program in Cardiovascular and Metabolic Disorders, Duke-NUS Medical School Singapore, Singapore.; National Heart Research Institute Singapore, National Heart Center Singapore, Singapore.
Source: JCI insight [JCI Insight] 2019 Aug 22; Vol. 4 (16). Date of Electronic Publication: 2019 Aug 22.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: American Society for Clinical Investigation Country of Publication: United States NLM ID: 101676073 Publication Model: Electronic Cited Medium: Internet ISSN: 2379-3708 (Electronic) Linking ISSN: 23793708 NLM ISO Abbreviation: JCI Insight Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Semaphorin 3E/PlexinD1 signaling is required for cardiac ventricular compaction.
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  Data: <searchLink fieldCode="AU" term="%22Sandireddy+R%22">Sandireddy R</searchLink>; Program in Cardiovascular and Metabolic Disorders, Duke-NUS Medical School Singapore, Singapore.<br /><searchLink fieldCode="AU" term="%22Cibi+DM%22">Cibi DM</searchLink>; Program in Cardiovascular and Metabolic Disorders, Duke-NUS Medical School Singapore, Singapore.<br /><searchLink fieldCode="AU" term="%22Gupta+P%22">Gupta P</searchLink>; Program in Cardiovascular and Metabolic Disorders, Duke-NUS Medical School Singapore, Singapore.<br /><searchLink fieldCode="AU" term="%22Singh+A%22">Singh A</searchLink>; Program in Cardiovascular and Metabolic Disorders, Duke-NUS Medical School Singapore, Singapore.<br /><searchLink fieldCode="AU" term="%22Tee+N%22">Tee N</searchLink>; National Heart Research Institute Singapore, National Heart Center Singapore, Singapore.<br /><searchLink fieldCode="AU" term="%22Uemura+A%22">Uemura A</searchLink>; Department of Retinal Vascular Biology, Nagoya City University Graduate School of Medical Sciences, Mizuho-ku, Nagoya, Japan.<br /><searchLink fieldCode="AU" term="%22Epstein+JA%22">Epstein JA</searchLink>; Penn Cardiovascular Institute, Department of Medicine, Department of Cell and Developmental Biology, and Institute for Regenerative Medicine, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, USA.<br /><searchLink fieldCode="AU" term="%22Singh+MK%22">Singh MK</searchLink>; Program in Cardiovascular and Metabolic Disorders, Duke-NUS Medical School Singapore, Singapore.; National Heart Research Institute Singapore, National Heart Center Singapore, Singapore.
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  Data: <searchLink fieldCode="JN" term="%22101676073%22">JCI insight</searchLink> [JCI Insight] 2019 Aug 22; Vol. 4 (16). <i>Date of Electronic Publication: </i>2019 Aug 22.
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        Value: 10.1172/jci.insight.125908
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      – TitleFull: Semaphorin 3E/PlexinD1 signaling is required for cardiac ventricular compaction.
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              Text: 2019 Aug 22
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              Y: 2019
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