Pharmacological inhibition of glycogen synthase kinase 3 increases operant alcohol self-administration in a manner associated with altered pGSK-3β, protein interacting with C kinase and GluA2 protein expression in the reward pathway of male C57BL/6J mice.
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| Title: | Pharmacological inhibition of glycogen synthase kinase 3 increases operant alcohol self-administration in a manner associated with altered pGSK-3β, protein interacting with C kinase and GluA2 protein expression in the reward pathway of male C57BL/6J mice. |
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| Authors: | Faccidomo S; Bowles Center for Alcohol Studies., Holstein SE; Bowles Center for Alcohol Studies., Santanam TS; Bowles Center for Alcohol Studies., Saunders BL; Bowles Center for Alcohol Studies., Swaim KS; Bowles Center for Alcohol Studies., Reid GT; Bowles Center for Alcohol Studies., O'Neill C; Bowles Center for Alcohol Studies., Eastman VR; Bowles Center for Alcohol Studies., Hodge CW; Bowles Center for Alcohol Studies.; Department of Psychiatry, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, USA. |
| Source: | Behavioural pharmacology [Behav Pharmacol] 2020 Feb; Vol. 31 (1), pp. 15-26. |
| Publication Type: | Journal Article; Research Support, N.I.H., Extramural |
| Journal Info: | Publisher: Lippincott Williams and Wilkins Country of Publication: England NLM ID: 9013016 Publication Model: Print Cited Medium: Internet ISSN: 1473-5849 (Electronic) Linking ISSN: 09558810 NLM ISO Abbreviation: Behav Pharmacol Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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