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Intermedin1-53 Ameliorates Homocysteine-Promoted Atherosclerotic Calcification by Inhibiting Endoplasmic Reticulum Stress.

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Title: Intermedin1-53 Ameliorates Homocysteine-Promoted Atherosclerotic Calcification by Inhibiting Endoplasmic Reticulum Stress.
Authors: Ren JL; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China., Hou YL; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China., Ni XQ; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China., Zhu Q; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China., Chen Y; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China., Zhang LS; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China., Liu X; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China., Xue CD; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China., Wu N; Department of Gynaecology and Obstetrics, Beijing Chao-Yang Hospital, Capital Medical University, Beijing, China., Yu YR; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China., Tang CS; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Peking University, Beijing, China., Ning ZP; Shanghai University of Medicine and Health Sciences, Shanghai University of Medicine and Health Sciences Affiliated Zhoupu Hospital, Shanghai, China., Chai SB; Department of Endocrinology, Peking University International Hospital, Beijing, China., Qi YF; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China.
Source: Journal of cardiovascular pharmacology and therapeutics [J Cardiovasc Pharmacol Ther] 2020 May; Vol. 25 (3), pp. 251-264. Date of Electronic Publication: 2019 Nov 07.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Sage Publications Country of Publication: United States NLM ID: 9602617 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1940-4034 (Electronic) Linking ISSN: 10742484 NLM ISO Abbreviation: J Cardiovasc Pharmacol Ther Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: <searchLink fieldCode="AU" term="%22Ren+JL%22">Ren JL</searchLink>; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Hou+YL%22">Hou YL</searchLink>; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Ni+XQ%22">Ni XQ</searchLink>; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Zhu+Q%22">Zhu Q</searchLink>; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Chen+Y%22">Chen Y</searchLink>; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Zhang+LS%22">Zhang LS</searchLink>; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Liu+X%22">Liu X</searchLink>; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Xue+CD%22">Xue CD</searchLink>; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Wu+N%22">Wu N</searchLink>; Department of Gynaecology and Obstetrics, Beijing Chao-Yang Hospital, Capital Medical University, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Yu+YR%22">Yu YR</searchLink>; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Tang+CS%22">Tang CS</searchLink>; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Peking University, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Ning+ZP%22">Ning ZP</searchLink>; Shanghai University of Medicine and Health Sciences, Shanghai University of Medicine and Health Sciences Affiliated Zhoupu Hospital, Shanghai, China.<br /><searchLink fieldCode="AU" term="%22Chai+SB%22">Chai SB</searchLink>; Department of Endocrinology, Peking University International Hospital, Beijing, China.<br /><searchLink fieldCode="AU" term="%22Qi+YF%22">Qi YF</searchLink>; Laboratory of Cardiovascular Bioactive Molecule, School of Basic Medical Sciences, Peking University, Beijing, China.; Key Laboratory of Molecular Cardiovascular Science, Ministry of Education, Peking University Health Science Center, Beijing, China.; Department of Pathogen Biology, School of Basic Medical Sciences, Peking University, Beijing, China.
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  Data: <searchLink fieldCode="JN" term="%229602617%22">Journal of cardiovascular pharmacology and therapeutics</searchLink> [J Cardiovasc Pharmacol Ther] 2020 May; Vol. 25 (3), pp. 251-264. <i>Date of Electronic Publication: </i>2019 Nov 07.
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      – TitleFull: Intermedin1-53 Ameliorates Homocysteine-Promoted Atherosclerotic Calcification by Inhibiting Endoplasmic Reticulum Stress.
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