Mitochondrial disease disrupts hepatic allostasis and lowers the threshold for immune-mediated liver toxicity.
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| Title: | Mitochondrial disease disrupts hepatic allostasis and lowers the threshold for immune-mediated liver toxicity. |
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| Authors: | Jestin M; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA., Kapnick SM; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA., Tarasenko TN; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA., Burke CT; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA., Zerfas PM; Office of Research Services, National Institutes of Health, Bethesda, MD, 20892, USA., Diaz F; University of Miami, Department of Neurology, Miller School of Medicine, Miami, FL, 33136, USA., Vernon H; Kennedy Krieger Institute, Johns Hopkins Medical Center, Baltimore, MD, 21205, USA., Singh LN; Center for Mitochondrial and Epigenomic Medicine, Children's Hospital of Philadelphia, Philadelphia, PA, 19104, USA., Sokol RJ; Section of Pediatric Gastroenterology, Hepatology and Nutrition, University of Colorado School of Medicine and Children's Hospital Colorado, Aurora, CO 80045, USA., McGuire PJ; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA. Electronic address: peter.mcguire@nih.gov. |
| Source: | Molecular metabolism [Mol Metab] 2020 Jul; Vol. 37, pp. 100981. Date of Electronic Publication: 2020 Mar 26. |
| Publication Type: | Journal Article; Research Support, N.I.H., Intramural |
| Journal Info: | Publisher: Elsevier GmbH Country of Publication: Germany NLM ID: 101605730 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2212-8778 (Electronic) Linking ISSN: 22128778 NLM ISO Abbreviation: Mol Metab Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
| FullText | Text: Availability: 0 |
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| Header | DbId: mdl DbLabel: MEDLINE Ultimate An: 32283081 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: Mitochondrial disease disrupts hepatic allostasis and lowers the threshold for immune-mediated liver toxicity. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AU" term="%22Jestin+M%22">Jestin M</searchLink>; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA.<br /><searchLink fieldCode="AU" term="%22Kapnick+SM%22">Kapnick SM</searchLink>; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA.<br /><searchLink fieldCode="AU" term="%22Tarasenko+TN%22">Tarasenko TN</searchLink>; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA.<br /><searchLink fieldCode="AU" term="%22Burke+CT%22">Burke CT</searchLink>; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA.<br /><searchLink fieldCode="AU" term="%22Zerfas+PM%22">Zerfas PM</searchLink>; Office of Research Services, National Institutes of Health, Bethesda, MD, 20892, USA.<br /><searchLink fieldCode="AU" term="%22Diaz+F%22">Diaz F</searchLink>; University of Miami, Department of Neurology, Miller School of Medicine, Miami, FL, 33136, USA.<br /><searchLink fieldCode="AU" term="%22Vernon+H%22">Vernon H</searchLink>; Kennedy Krieger Institute, Johns Hopkins Medical Center, Baltimore, MD, 21205, USA.<br /><searchLink fieldCode="AU" term="%22Singh+LN%22">Singh LN</searchLink>; Center for Mitochondrial and Epigenomic Medicine, Children's Hospital of Philadelphia, Philadelphia, PA, 19104, USA.<br /><searchLink fieldCode="AU" term="%22Sokol+RJ%22">Sokol RJ</searchLink>; Section of Pediatric Gastroenterology, Hepatology and Nutrition, University of Colorado School of Medicine and Children's Hospital Colorado, Aurora, CO 80045, USA.<br /><searchLink fieldCode="AU" term="%22McGuire+PJ%22">McGuire PJ</searchLink>; National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, 20892, USA. Electronic address: peter.mcguire@nih.gov. – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%22101605730%22">Molecular metabolism</searchLink> [Mol Metab] 2020 Jul; Vol. 37, pp. 100981. <i>Date of Electronic Publication: </i>2020 Mar 26. – Name: TypePub Label: Publication Type Group: TypPub Data: Journal Article; Research Support, N.I.H., Intramural – Name: TitleSource Label: Journal Info Group: Src Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Elsevier+GmbH%22">Elsevier GmbH </searchLink><i>Country of Publication: </i>Germany <i>NLM ID: </i>101605730 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>2212-8778 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2222128778%22">22128778 </searchLink><i>NLM ISO Abbreviation: </i>Mol Metab <i>Subsets: </i>MEDLINE |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=32283081 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.1016/j.molmet.2020.100981 Languages: – Code: eng Text: English PhysicalDescription: Pagination: StartPage: 100981 Titles: – TitleFull: Mitochondrial disease disrupts hepatic allostasis and lowers the threshold for immune-mediated liver toxicity. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Jestin M – PersonEntity: Name: NameFull: Kapnick SM – PersonEntity: Name: NameFull: Tarasenko TN – PersonEntity: Name: NameFull: Burke CT – PersonEntity: Name: NameFull: Zerfas PM – PersonEntity: Name: NameFull: Diaz F – PersonEntity: Name: NameFull: Vernon H – PersonEntity: Name: NameFull: Singh LN – PersonEntity: Name: NameFull: Sokol RJ – PersonEntity: Name: NameFull: McGuire PJ IsPartOfRelationships: – BibEntity: Dates: – D: 01 M: 07 Text: 2020 Jul Type: published Y: 2020 Identifiers: – Type: issn-electronic Value: 2212-8778 Numbering: – Type: volume Value: 37 Titles: – TitleFull: Molecular metabolism Type: main |
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