Spleen Tyrosine Kinase Is a Critical Regulator of Neutrophil Responses to Candida Species.

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Bibliographic Details
Title: Spleen Tyrosine Kinase Is a Critical Regulator of Neutrophil Responses to Candida Species.
Authors: Negoro PE; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA., Xu S; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA., Dagher Z; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA., Hopke A; Department of Surgery, Massachusetts General Hospital, Boston, Massachusetts, USA.; Harvard Medical School, Boston, Massachusetts, USA., Reedy JL; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA.; Harvard Medical School, Boston, Massachusetts, USA., Feldman MB; Division of Pulmonary and Critical Care Medicine, Massachusetts General Hospital, Boston Massachusetts, USA.; Harvard Medical School, Boston, Massachusetts, USA., Khan NS; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA.; Harvard Medical School, Boston, Massachusetts, USA., Viens AL; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA., Alexander NJ; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA., Atallah NJ; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA., Scherer AK; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA.; Harvard Medical School, Boston, Massachusetts, USA., Dutko RA; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA., Jeffery J; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA., Kernien JF; Department of Medicine, University of Wisconsin-Madison, Madison Wisconsin, USA., Fites JS; Department of Pediatrics, University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin, USA., Nett JE; Department of Medical Microbiology and Immunology, University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin, USA.; Department of Medicine, University of Wisconsin-Madison, Madison Wisconsin, USA., Klein BS; Department of Medical Microbiology and Immunology, University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin, USA.; Department of Pediatrics, University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin, USA.; Department of Medicine, University of Wisconsin-Madison, Madison Wisconsin, USA., Vyas JM; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA.; Harvard Medical School, Boston, Massachusetts, USA., Irimia D; Department of Surgery, Massachusetts General Hospital, Boston, Massachusetts, USA.; Harvard Medical School, Boston, Massachusetts, USA., Sykes DB; Center for Regenerative Medicine, Massachusetts General Hospital, Boston, Massachusetts, USA.; Harvard Medical School, Boston, Massachusetts, USA., Mansour MK; Division of Infectious Diseases, Massachusetts General Hospital, Boston, Massachusetts, USA mkmansour@mgh.harvard.edu.; Harvard Medical School, Boston, Massachusetts, USA.
Source: MBio [mBio] 2020 May 12; Vol. 11 (3). Date of Electronic Publication: 2020 May 12.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: American Society for Microbiology Country of Publication: United States NLM ID: 101519231 Publication Model: Electronic Cited Medium: Internet ISSN: 2150-7511 (Electronic) NLM ISO Abbreviation: mBio Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:2150-7511
DOI:10.1128/mBio.02043-19