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Metabolic excretion associated with nutrient-growth dysregulation promotes the rapid evolution of an overt metabolic defect.

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Title: Metabolic excretion associated with nutrient-growth dysregulation promotes the rapid evolution of an overt metabolic defect.
Authors: Green R; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America., Sonal; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America., Wang L; Department of Chemistry and Lewis-Sigler Institute for Integrative Genomics, Princeton University, Princeton, New Jersey, United States of America., Hart SFM; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America., Lu W; Department of Chemistry and Lewis-Sigler Institute for Integrative Genomics, Princeton University, Princeton, New Jersey, United States of America., Skelding D; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America., Burton JC; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America., Mi H; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America., Capel A; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America., Chen HA; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America., Lin A; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America., Subramaniam AR; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America., Rabinowitz JD; Department of Chemistry and Lewis-Sigler Institute for Integrative Genomics, Princeton University, Princeton, New Jersey, United States of America., Shou W; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America.
Source: PLoS biology [PLoS Biol] 2020 Aug 24; Vol. 18 (8), pp. e3000757. Date of Electronic Publication: 2020 Aug 24 (Print Publication: 2020).
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
Journal Info: Publisher: Public Library of Science Country of Publication: United States NLM ID: 101183755 Publication Model: eCollection Cited Medium: Internet ISSN: 1545-7885 (Electronic) Linking ISSN: 15449173 NLM ISO Abbreviation: PLoS Biol Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Metabolic excretion associated with nutrient-growth dysregulation promotes the rapid evolution of an overt metabolic defect.
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  Data: <searchLink fieldCode="AU" term="%22Green+R%22">Green R</searchLink>; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America.<br /><searchLink fieldCode="AU" term="%22Sonal%22">Sonal</searchLink>; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America.<br /><searchLink fieldCode="AU" term="%22Wang+L%22">Wang L</searchLink>; Department of Chemistry and Lewis-Sigler Institute for Integrative Genomics, Princeton University, Princeton, New Jersey, United States of America.<br /><searchLink fieldCode="AU" term="%22Hart+SFM%22">Hart SFM</searchLink>; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America.<br /><searchLink fieldCode="AU" term="%22Lu+W%22">Lu W</searchLink>; Department of Chemistry and Lewis-Sigler Institute for Integrative Genomics, Princeton University, Princeton, New Jersey, United States of America.<br /><searchLink fieldCode="AU" term="%22Skelding+D%22">Skelding D</searchLink>; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America.<br /><searchLink fieldCode="AU" term="%22Burton+JC%22">Burton JC</searchLink>; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America.<br /><searchLink fieldCode="AU" term="%22Mi+H%22">Mi H</searchLink>; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America.<br /><searchLink fieldCode="AU" term="%22Capel+A%22">Capel A</searchLink>; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America.<br /><searchLink fieldCode="AU" term="%22Chen+HA%22">Chen HA</searchLink>; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America.<br /><searchLink fieldCode="AU" term="%22Lin+A%22">Lin A</searchLink>; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America.<br /><searchLink fieldCode="AU" term="%22Subramaniam+AR%22">Subramaniam AR</searchLink>; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America.<br /><searchLink fieldCode="AU" term="%22Rabinowitz+JD%22">Rabinowitz JD</searchLink>; Department of Chemistry and Lewis-Sigler Institute for Integrative Genomics, Princeton University, Princeton, New Jersey, United States of America.<br /><searchLink fieldCode="AU" term="%22Shou+W%22">Shou W</searchLink>; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, Washington, United States of America.
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  Data: <searchLink fieldCode="JN" term="%22101183755%22">PLoS biology</searchLink> [PLoS Biol] 2020 Aug 24; Vol. 18 (8), pp. e3000757. <i>Date of Electronic Publication: </i>2020 Aug 24 (<i>Print Publication: </i>2020).
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