Kv1.1 potassium channel subunit deficiency alters ventricular arrhythmia susceptibility, contractility, and repolarization.

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Bibliographic Details
Title: Kv1.1 potassium channel subunit deficiency alters ventricular arrhythmia susceptibility, contractility, and repolarization.
Authors: Trosclair K; Department of Cellular Biology & Anatomy, Louisiana State University Health Sciences Center, Shreveport, LA, USA., Si M; Department of Cellular Biology & Anatomy, Louisiana State University Health Sciences Center, Shreveport, LA, USA., Watts M; Department of Internal Medicine, Section of Cardiology, Louisiana State University Health Sciences Center, Shreveport, LA, USA., Gautier NM; Department of Cellular Biology & Anatomy, Louisiana State University Health Sciences Center, Shreveport, LA, USA., Voigt N; Institute of Pharmacology and Toxicology, University Medical Center Goettingen, Goettingen, Germany.; DZHK (German Center for Cardiovascular Research), Göttingen, Germany., Traylor J; Department of Pathology, Louisiana State University Health Sciences Center, Shreveport, LA, USA., Bitay M; Department of Cardiac Surgery, 2nd Department of Medicine and Cardiology Center, University of Szeged, Szeged, Hungary., Baczko I; Department of Pharmacology and Pharmacotherapy, Interdisciplinary Excellence Centre, University of Szeged, Szeged, Hungary., Dobrev D; Institute of Pharmacology, West German Heart and Vascular Center, University Duisburg-Essen, Essen, Germany., Hamilton KA; Department of Cellular Biology & Anatomy, Louisiana State University Health Sciences Center, Shreveport, LA, USA., Bhuiyan MS; Department of Pathology, Louisiana State University Health Sciences Center, Shreveport, LA, USA., Dominic P; Department of Internal Medicine, Section of Cardiology, Louisiana State University Health Sciences Center, Shreveport, LA, USA., Glasscock E; Department of Cellular Biology & Anatomy, Louisiana State University Health Sciences Center, Shreveport, LA, USA.; Department of Biological Sciences, Southern Methodist University, Dallas, TX, USA.
Source: Physiological reports [Physiol Rep] 2021 Jan; Vol. 9 (1), pp. e14702.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: published by Wiley Periodicals, Inc. on behalf of the American Physiological Society and The Physiological Society Country of Publication: United States NLM ID: 101607800 Publication Model: Print Cited Medium: Internet ISSN: 2051-817X (Electronic) Linking ISSN: 2051817X NLM ISO Abbreviation: Physiol Rep Subsets: MEDLINE
Database: MEDLINE Ultimate
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