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Role of the transcriptional regulator SP140 in resistance to bacterial infections via repression of type I interferons.

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Title: Role of the transcriptional regulator SP140 in resistance to bacterial infections via repression of type I interferons.
Authors: Ji DX; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States., Witt KC; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States., Kotov DI; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States.; Howard Hughes Medical Institute, University of California, Berkeley, Berkeley, United States., Margolis SR; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States., Louie A; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States., Chevée V; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States., Chen KJ; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States.; Howard Hughes Medical Institute, University of California, Berkeley, Berkeley, United States., Gaidt MM; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States., Dhaliwal HS; Cancer Research Laboratory, University of California, Berkeley, Berkeley, United States., Lee AY; Cancer Research Laboratory, University of California, Berkeley, Berkeley, United States., Nishimura SL; Department of Pathology, University of California, San Francisco, San Francisco, United States., Zamboni DS; Department of Cell Biology, Ribeirão Preto Medical School, University of São Paulo, São Paulo, Brazil., Kramnik I; The National Emerging Infectious Diseases Laboratory, Department of Medicine (Pulmonary Center), and Department of Microbiology, Boston University School of Medicine, Boston, United States., Portnoy DA; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States.; Division of Biochemistry, Biophysics and Structural Biology, Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States.; Department of Plant and Microbial Biology, University of California, Berkeley, Berkeley, United States., Darwin KH; Department of Microbiology, New York University Grossman School of Medicine, New York, United States., Vance RE; Division of Immunology and Pathogenesis, Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States.; Howard Hughes Medical Institute, University of California, Berkeley, Berkeley, United States.; Cancer Research Laboratory, University of California, Berkeley, Berkeley, United States.
Source: ELife [Elife] 2021 Jun 21; Vol. 10. Date of Electronic Publication: 2021 Jun 21.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: eLife Sciences Publications, Ltd Country of Publication: England NLM ID: 101579614 Publication Model: Electronic Cited Medium: Internet ISSN: 2050-084X (Electronic) Linking ISSN: 2050084X NLM ISO Abbreviation: Elife Subsets: MEDLINE
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  Data: Role of the transcriptional regulator SP140 in resistance to bacterial infections via repression of type I interferons.
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