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Pharmacological and genetic manipulations at the µ-opioid receptor reveal arrestin-3 engagement limits analgesic tolerance and does not exacerbate respiratory depression in mice.

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Title: Pharmacological and genetic manipulations at the µ-opioid receptor reveal arrestin-3 engagement limits analgesic tolerance and does not exacerbate respiratory depression in mice.
Authors: He L; Center for Neuroscience, University of California-Davis, Davis, CA, USA.; Department of Physiology and Membrane Biology, UC Davis School of Medicine, Davis, CA, USA., Gooding SW; Center for Neuroscience, University of California-Davis, Davis, CA, USA., Lewis E; Center for Neuroscience, University of California-Davis, Davis, CA, USA., Felth LC; Center for Neuroscience, University of California-Davis, Davis, CA, USA., Gaur A; Center for Neuroscience, University of California-Davis, Davis, CA, USA., Whistler JL; Center for Neuroscience, University of California-Davis, Davis, CA, USA. jlwhistler@ucdavis.edu.; Department of Physiology and Membrane Biology, UC Davis School of Medicine, Davis, CA, USA. jlwhistler@ucdavis.edu.
Source: Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology [Neuropsychopharmacology] 2021 Dec; Vol. 46 (13), pp. 2241-2249. Date of Electronic Publication: 2021 Jul 13.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Nature Publishing Group Country of Publication: England NLM ID: 8904907 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1740-634X (Electronic) Linking ISSN: 0893133X NLM ISO Abbreviation: Neuropsychopharmacology Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Pharmacological and genetic manipulations at the µ-opioid receptor reveal arrestin-3 engagement limits analgesic tolerance and does not exacerbate respiratory depression in mice.
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  Data: <searchLink fieldCode="AU" term="%22He+L%22">He L</searchLink>; Center for Neuroscience, University of California-Davis, Davis, CA, USA.; Department of Physiology and Membrane Biology, UC Davis School of Medicine, Davis, CA, USA.<br /><searchLink fieldCode="AU" term="%22Gooding+SW%22">Gooding SW</searchLink>; Center for Neuroscience, University of California-Davis, Davis, CA, USA.<br /><searchLink fieldCode="AU" term="%22Lewis+E%22">Lewis E</searchLink>; Center for Neuroscience, University of California-Davis, Davis, CA, USA.<br /><searchLink fieldCode="AU" term="%22Felth+LC%22">Felth LC</searchLink>; Center for Neuroscience, University of California-Davis, Davis, CA, USA.<br /><searchLink fieldCode="AU" term="%22Gaur+A%22">Gaur A</searchLink>; Center for Neuroscience, University of California-Davis, Davis, CA, USA.<br /><searchLink fieldCode="AU" term="%22Whistler+JL%22">Whistler JL</searchLink>; Center for Neuroscience, University of California-Davis, Davis, CA, USA. jlwhistler@ucdavis.edu.; Department of Physiology and Membrane Biology, UC Davis School of Medicine, Davis, CA, USA. jlwhistler@ucdavis.edu.
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  Data: <searchLink fieldCode="JN" term="%228904907%22">Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology</searchLink> [Neuropsychopharmacology] 2021 Dec; Vol. 46 (13), pp. 2241-2249. <i>Date of Electronic Publication: </i>2021 Jul 13.
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        Value: 10.1038/s41386-021-01054-x
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