Targeted deletion of PAC1 receptors in retinal neurons enhances neuron loss and axonopathy in a model of multiple sclerosis and optic neuritis.

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Title: Targeted deletion of PAC1 receptors in retinal neurons enhances neuron loss and axonopathy in a model of multiple sclerosis and optic neuritis.
Authors: Van C; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America; Molecular Biology Interdepartmental Program at University of California, Los Angeles, Los Angeles, CA 90095, United States of America. Electronic address: christinavan@ucla.edu., Condro MC; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America. Electronic address: mcondro@mednet.ucla.edu., Ko HH; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America., Hoang AQ; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America. Electronic address: ahoang1993@g.ucla.edu., Zhu R; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America. Electronic address: zhur@email.arizona.edu., Lov K; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America., Ricaflanca PT; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America., Diep AL; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America. Electronic address: diepal@hs.uci.edu., Nguyen NNM; Calabasas High School, Calabasas, CA 91302, United States of America. Electronic address: Nhat.Nguyen@ucsf.edu., Lipshutz GS; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America; Molecular Biology Interdepartmental Program at University of California, Los Angeles, Los Angeles, CA 90095, United States of America; Departments of Surgery, Medical Pharmacology, Urology, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, United States of America. Electronic address: glipshutz@mednet.ucla.edu., MacKenzie-Graham A; Department of Neurology, David Geffen School of Medicine, University of California, Los Angeles, CA 90095, United States of America. Electronic address: AMackenzie@mednet.ucla.edu., Waschek JA; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America. Electronic address: jwaschek@mednet.ucla.edu.
Source: Neurobiology of disease [Neurobiol Dis] 2021 Dec; Vol. 160, pp. 105524. Date of Electronic Publication: 2021 Oct 02.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
Journal Info: Publisher: Academic Press Country of Publication: United States NLM ID: 9500169 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1095-953X (Electronic) Linking ISSN: 09699961 NLM ISO Abbreviation: Neurobiol Dis Subsets: MEDLINE
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  Data: Targeted deletion of PAC1 receptors in retinal neurons enhances neuron loss and axonopathy in a model of multiple sclerosis and optic neuritis.
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  Data: <searchLink fieldCode="AU" term="%22Van+C%22">Van C</searchLink>; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America; Molecular Biology Interdepartmental Program at University of California, Los Angeles, Los Angeles, CA 90095, United States of America. Electronic address: christinavan@ucla.edu.<br /><searchLink fieldCode="AU" term="%22Condro+MC%22">Condro MC</searchLink>; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America. Electronic address: mcondro@mednet.ucla.edu.<br /><searchLink fieldCode="AU" term="%22Ko+HH%22">Ko HH</searchLink>; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America.<br /><searchLink fieldCode="AU" term="%22Hoang+AQ%22">Hoang AQ</searchLink>; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America. Electronic address: ahoang1993@g.ucla.edu.<br /><searchLink fieldCode="AU" term="%22Zhu+R%22">Zhu R</searchLink>; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America. Electronic address: zhur@email.arizona.edu.<br /><searchLink fieldCode="AU" term="%22Lov+K%22">Lov K</searchLink>; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America.<br /><searchLink fieldCode="AU" term="%22Ricaflanca+PT%22">Ricaflanca PT</searchLink>; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America.<br /><searchLink fieldCode="AU" term="%22Diep+AL%22">Diep AL</searchLink>; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America. Electronic address: diepal@hs.uci.edu.<br /><searchLink fieldCode="AU" term="%22Nguyen+NNM%22">Nguyen NNM</searchLink>; Calabasas High School, Calabasas, CA 91302, United States of America. Electronic address: Nhat.Nguyen@ucsf.edu.<br /><searchLink fieldCode="AU" term="%22Lipshutz+GS%22">Lipshutz GS</searchLink>; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America; Molecular Biology Interdepartmental Program at University of California, Los Angeles, Los Angeles, CA 90095, United States of America; Departments of Surgery, Medical Pharmacology, Urology, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, United States of America. Electronic address: glipshutz@mednet.ucla.edu.<br /><searchLink fieldCode="AU" term="%22MacKenzie-Graham+A%22">MacKenzie-Graham A</searchLink>; Department of Neurology, David Geffen School of Medicine, University of California, Los Angeles, CA 90095, United States of America. Electronic address: AMackenzie@mednet.ucla.edu.<br /><searchLink fieldCode="AU" term="%22Waschek+JA%22">Waschek JA</searchLink>; Semel Institute for Neuroscience and Human Behavior/Neuropsychiatric Institute, Department of Psychiatry, Intellectual and Developmental Disabilities Research Center, University of California, Los Angeles, Los Angeles, CA 90095, United States of America. Electronic address: jwaschek@mednet.ucla.edu.
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  Data: <searchLink fieldCode="JN" term="%229500169%22">Neurobiology of disease</searchLink> [Neurobiol Dis] 2021 Dec; Vol. 160, pp. 105524. <i>Date of Electronic Publication: </i>2021 Oct 02.
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        Value: 10.1016/j.nbd.2021.105524
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        Text: English
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      – TitleFull: Targeted deletion of PAC1 receptors in retinal neurons enhances neuron loss and axonopathy in a model of multiple sclerosis and optic neuritis.
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              Text: 2021 Dec
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