Copper-imidazo[1,2-a]pyridines differentially modulate pro- and anti-apoptotic protein and gene expression in HL-60 and K562 leukaemic cells to cause apoptotic cell death.

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Title: Copper-imidazo[1,2-a]pyridines differentially modulate pro- and anti-apoptotic protein and gene expression in HL-60 and K562 leukaemic cells to cause apoptotic cell death.
Authors: Ismail Z; Division of Pharmacology, Department of Pharmacy and Pharmacology, School of Therapeutic Sciences, Faculty of Health Sciences, University of the Witwatersrand, 7 York Road, Parktown 2193, South Africa., Dam J; Molecular Sciences Institute, School of Chemistry, University of the Witwatersrand, PO Wits 2050, South Africa., Penny C; Department of Internal Medicine, Faculty of Health Sciences, University of the Witwatersrand, 7 York Road, Parktown 2193, South Africa., de Koning CB; Molecular Sciences Institute, School of Chemistry, University of the Witwatersrand, PO Wits 2050, South Africa., Harmse L; Division of Pharmacology, Department of Pharmacy and Pharmacology, School of Therapeutic Sciences, Faculty of Health Sciences, University of the Witwatersrand, 7 York Road, Parktown 2193, South Africa. Electronic address: Leonie.Harmse@wits.ac.za.
Source: Biochimica et biophysica acta. Molecular cell research [Biochim Biophys Acta Mol Cell Res] 2022 Jan; Vol. 1869 (1), pp. 119160. Date of Electronic Publication: 2021 Oct 09.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Elsevier Country of Publication: Netherlands NLM ID: 101731731 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1879-2596 (Electronic) Linking ISSN: 01674889 NLM ISO Abbreviation: Biochim Biophys Acta Mol Cell Res Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Copper-imidazo[1,2-a]pyridines differentially modulate pro- and anti-apoptotic protein and gene expression in HL-60 and K562 leukaemic cells to cause apoptotic cell death.
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  Data: <searchLink fieldCode="AU" term="%22Ismail+Z%22">Ismail Z</searchLink>; Division of Pharmacology, Department of Pharmacy and Pharmacology, School of Therapeutic Sciences, Faculty of Health Sciences, University of the Witwatersrand, 7 York Road, Parktown 2193, South Africa.<br /><searchLink fieldCode="AU" term="%22Dam+J%22">Dam J</searchLink>; Molecular Sciences Institute, School of Chemistry, University of the Witwatersrand, PO Wits 2050, South Africa.<br /><searchLink fieldCode="AU" term="%22Penny+C%22">Penny C</searchLink>; Department of Internal Medicine, Faculty of Health Sciences, University of the Witwatersrand, 7 York Road, Parktown 2193, South Africa.<br /><searchLink fieldCode="AU" term="%22de+Koning+CB%22">de Koning CB</searchLink>; Molecular Sciences Institute, School of Chemistry, University of the Witwatersrand, PO Wits 2050, South Africa.<br /><searchLink fieldCode="AU" term="%22Harmse+L%22">Harmse L</searchLink>; Division of Pharmacology, Department of Pharmacy and Pharmacology, School of Therapeutic Sciences, Faculty of Health Sciences, University of the Witwatersrand, 7 York Road, Parktown 2193, South Africa. Electronic address: Leonie.Harmse@wits.ac.za.
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  Data: <searchLink fieldCode="JN" term="%22101731731%22">Biochimica et biophysica acta. Molecular cell research</searchLink> [Biochim Biophys Acta Mol Cell Res] 2022 Jan; Vol. 1869 (1), pp. 119160. <i>Date of Electronic Publication: </i>2021 Oct 09.
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        Value: 10.1016/j.bbamcr.2021.119160
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      – TitleFull: Copper-imidazo[1,2-a]pyridines differentially modulate pro- and anti-apoptotic protein and gene expression in HL-60 and K562 leukaemic cells to cause apoptotic cell death.
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              Text: 2022 Jan
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