The nuclear receptor REV-ERBα is implicated in the alteration of β-cell autophagy and survival under diabetogenic conditions.

Saved in:
Bibliographic Details
Title: The nuclear receptor REV-ERBα is implicated in the alteration of β-cell autophagy and survival under diabetogenic conditions.
Authors: Brown MR; Department of Physiology and Biomedical Engineering, Mayo Clinic School of Medicine, Mayo Clinic, Rochester, MN, USA., Laouteouet D; Institute of Functional Genomics, University of Montpellier, CNRS, INSERM, Montpellier, France., Delobel M; Institute of Functional Genomics, University of Montpellier, CNRS, INSERM, Montpellier, France., Villard O; Laboratory of Cell Therapy for Diabetes (LTCD), PRIMS facility, Institute for Regenerative Medicine and Biotherapy (IRMB), University hospital of Montpellier, Montpellier, France.; Department of Endocrinology, Diabetes, and Nutrition, University Hospital of Montpellier, Montpellier, France., Broca C; Laboratory of Cell Therapy for Diabetes (LTCD), PRIMS facility, Institute for Regenerative Medicine and Biotherapy (IRMB), University hospital of Montpellier, Montpellier, France., Bertrand G; Institute of Functional Genomics, University of Montpellier, CNRS, INSERM, Montpellier, France., Wojtusciszyn A; Institute of Functional Genomics, University of Montpellier, CNRS, INSERM, Montpellier, France.; Laboratory of Cell Therapy for Diabetes (LTCD), PRIMS facility, Institute for Regenerative Medicine and Biotherapy (IRMB), University hospital of Montpellier, Montpellier, France.; Department of Endocrinology, Diabetes, and Nutrition, University Hospital of Montpellier, Montpellier, France., Dalle S; Institute of Functional Genomics, University of Montpellier, CNRS, INSERM, Montpellier, France., Ravier MA; Institute of Functional Genomics, University of Montpellier, CNRS, INSERM, Montpellier, France., Matveyenko AV; Department of Physiology and Biomedical Engineering, Mayo Clinic School of Medicine, Mayo Clinic, Rochester, MN, USA. Matveyenko.Aleksey@mayo.edu.; Division of Endocrinology, Metabolism, Diabetes, and Nutrition, Department of Medicine, Mayo Clinic, Rochester, MN, USA. Matveyenko.Aleksey@mayo.edu., Costes S; Institute of Functional Genomics, University of Montpellier, CNRS, INSERM, Montpellier, France. safia.costes@igf.cnrs.fr.
Source: Cell death & disease [Cell Death Dis] 2022 Apr 15; Vol. 13 (4), pp. 353. Date of Electronic Publication: 2022 Apr 15.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
Journal Info: Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101524092 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-4889 (Electronic) NLM ISO Abbreviation: Cell Death Dis Subsets: MEDLINE
Database: MEDLINE Ultimate
Full text is not displayed to guests.
Description
ISSN:2041-4889
DOI:10.1038/s41419-022-04767-z