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Hydroxyproline stimulates inflammation and reprograms macrophage signaling in a rat kidney stone model.

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Title: Hydroxyproline stimulates inflammation and reprograms macrophage signaling in a rat kidney stone model.
Authors: Kumar P; Department of Urology, University of Alabama Birmingham, Birmingham, AL, USA., Yang Z; Department of Nephrology, University of Alabama Birmingham, Birmingham, AL, USA., Lever JM; Department of Nephrology, University of Alabama Birmingham, Birmingham, AL, USA., Chávez MD; Department of Urology, University of Alabama Birmingham, Birmingham, AL, USA., Fatima H; Department of Pathology, University of Alabama Birmingham, Birmingham, AL, USA., Crossman DK; Department of Medicine, University of Alabama Birmingham, Birmingham, AL, USA., Maynard CL; Department of Pathology, University of Alabama Birmingham, Birmingham, AL, USA., George JF; Department of Nephrology, University of Alabama Birmingham, Birmingham, AL, USA., Mitchell T; Department of Urology, University of Alabama Birmingham, Birmingham, AL, USA. Electronic address: taneciamitchell@uabmc.edu.
Source: Biochimica et biophysica acta. Molecular basis of disease [Biochim Biophys Acta Mol Basis Dis] 2022 Sep 01; Vol. 1868 (9), pp. 166442. Date of Electronic Publication: 2022 May 10.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
Journal Info: Publisher: Elsevier Country of Publication: Netherlands NLM ID: 101731730 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1879-260X (Electronic) Linking ISSN: 09254439 NLM ISO Abbreviation: Biochim Biophys Acta Mol Basis Dis Subsets: MEDLINE
Database: MEDLINE Ultimate
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PubType: Academic Journal
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  Data: Hydroxyproline stimulates inflammation and reprograms macrophage signaling in a rat kidney stone model.
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  Data: <searchLink fieldCode="AU" term="%22Kumar+P%22">Kumar P</searchLink>; Department of Urology, University of Alabama Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Yang+Z%22">Yang Z</searchLink>; Department of Nephrology, University of Alabama Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Lever+JM%22">Lever JM</searchLink>; Department of Nephrology, University of Alabama Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Chávez+MD%22">Chávez MD</searchLink>; Department of Urology, University of Alabama Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Fatima+H%22">Fatima H</searchLink>; Department of Pathology, University of Alabama Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Crossman+DK%22">Crossman DK</searchLink>; Department of Medicine, University of Alabama Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Maynard+CL%22">Maynard CL</searchLink>; Department of Pathology, University of Alabama Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22George+JF%22">George JF</searchLink>; Department of Nephrology, University of Alabama Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Mitchell+T%22">Mitchell T</searchLink>; Department of Urology, University of Alabama Birmingham, Birmingham, AL, USA. Electronic address: taneciamitchell@uabmc.edu.
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  Data: <searchLink fieldCode="JN" term="%22101731730%22">Biochimica et biophysica acta. Molecular basis of disease</searchLink> [Biochim Biophys Acta Mol Basis Dis] 2022 Sep 01; Vol. 1868 (9), pp. 166442. <i>Date of Electronic Publication: </i>2022 May 10.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Elsevier%22">Elsevier </searchLink><i>Country of Publication: </i>Netherlands <i>NLM ID: </i>101731730 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1879-260X (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2209254439%22">09254439 </searchLink><i>NLM ISO Abbreviation: </i>Biochim Biophys Acta Mol Basis Dis <i>Subsets: </i>MEDLINE
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        Value: 10.1016/j.bbadis.2022.166442
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        Text: English
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      – TitleFull: Hydroxyproline stimulates inflammation and reprograms macrophage signaling in a rat kidney stone model.
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              M: 09
              Text: 2022 Sep 01
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