β Cell-specific deletion of Zfp148 improves nutrient-stimulated β cell Ca2+ responses.
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| Title: | β Cell-specific deletion of Zfp148 improves nutrient-stimulated β cell Ca2+ responses. |
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| Authors: | Emfinger CH; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, USA., de Klerk E; UCSF Diabetes Center, UCSF, San Francisco, California, USA., Schueler KL; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, USA., Rabaglia ME; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, USA., Stapleton DS; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, USA., Simonett SP; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, USA., Mitok KA; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, USA., Wang Z; Department of Biostatistics and Medical Informatics, University of Wisconsin-Madison, Madison, Wisconsin, USA.; Biostatistics and Computational Biology Branch, National Institute of Environmental Health Sciences, Durham, North Carolina, USA., Liu X; Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA., Paulo JA; Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA., Yu Q; Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA., Cardone RL; Department of Internal Medicine (Endocrinology), Yale University, New Haven, Connecticut, USA., Foster HR; Department of Medicine, Division of Endocrinology, University of Wisconsin-Madison, Madison, Wisconsin, USA., Lewandowski SL; Department of Medicine, Division of Endocrinology, University of Wisconsin-Madison, Madison, Wisconsin, USA., Perales JC; Department of Physiological Sciences, School of Medicine, University of Barcelona, L'Hospitalet del Llobregat, Barcelona, Spain., Kendziorski CM; Department of Biostatistics and Medical Informatics, University of Wisconsin-Madison, Madison, Wisconsin, USA., Gygi SP; Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA., Kibbey RG; Department of Internal Medicine (Endocrinology), Yale University, New Haven, Connecticut, USA.; Department of Cellular and Molecular Physiology, Yale University, New Haven, Connecticut, USA., Keller MP; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, USA., Hebrok M; UCSF Diabetes Center, UCSF, San Francisco, California, USA., Merrins MJ; Department of Medicine, Division of Endocrinology, University of Wisconsin-Madison, Madison, Wisconsin, USA.; William S. Middleton Memorial Veterans Hospital, Madison, Wisconsin, USA., Attie AD; Department of Biochemistry, University of Wisconsin-Madison, Madison, Wisconsin, USA. |
| Source: | JCI insight [JCI Insight] 2022 May 23; Vol. 7 (10). Date of Electronic Publication: 2022 May 23. |
| Publication Type: | Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S. |
| Journal Info: | Publisher: American Society for Clinical Investigation Country of Publication: United States NLM ID: 101676073 Publication Model: Electronic Cited Medium: Internet ISSN: 2379-3708 (Electronic) Linking ISSN: 23793708 NLM ISO Abbreviation: JCI Insight Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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