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Endothelin receptor antagonism improves glucose tolerance and adipose tissue inflammation in an experimental model of systemic lupus erythematosus.

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Title: Endothelin receptor antagonism improves glucose tolerance and adipose tissue inflammation in an experimental model of systemic lupus erythematosus.
Authors: Rivera-Gonzalez O; Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi., Case CT; Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi., Wilson NA; Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi., Speed JS; Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi., Taylor EB; Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi.
Source: American journal of physiology. Endocrinology and metabolism [Am J Physiol Endocrinol Metab] 2023 Jan 01; Vol. 324 (1), pp. E73-E84. Date of Electronic Publication: 2022 Dec 07.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: American Physiological Society Country of Publication: United States NLM ID: 100901226 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-1555 (Electronic) Linking ISSN: 01931849 NLM ISO Abbreviation: Am J Physiol Endocrinol Metab Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Endothelin receptor antagonism improves glucose tolerance and adipose tissue inflammation in an experimental model of systemic lupus erythematosus.
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  Data: <searchLink fieldCode="AU" term="%22Rivera-Gonzalez+O%22">Rivera-Gonzalez O</searchLink>; Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi.<br /><searchLink fieldCode="AU" term="%22Case+CT%22">Case CT</searchLink>; Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi.<br /><searchLink fieldCode="AU" term="%22Wilson+NA%22">Wilson NA</searchLink>; Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi.<br /><searchLink fieldCode="AU" term="%22Speed+JS%22">Speed JS</searchLink>; Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi.<br /><searchLink fieldCode="AU" term="%22Taylor+EB%22">Taylor EB</searchLink>; Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi.
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  Data: <searchLink fieldCode="JN" term="%22100901226%22">American journal of physiology. Endocrinology and metabolism</searchLink> [Am J Physiol Endocrinol Metab] 2023 Jan 01; Vol. 324 (1), pp. E73-E84. <i>Date of Electronic Publication: </i>2022 Dec 07.
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  Data: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22American+Physiological+Society%22">American Physiological Society </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>100901226 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1522-1555 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2201931849%22">01931849 </searchLink><i>NLM ISO Abbreviation: </i>Am J Physiol Endocrinol Metab <i>Subsets: </i>MEDLINE
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        Value: 10.1152/ajpendo.00274.2022
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        Text: English
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              Text: 2023 Jan 01
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