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Autophagy and protein aggregation as a mechanism of dopaminergic degeneration in a primary human dopaminergic neuronal model.

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Title: Autophagy and protein aggregation as a mechanism of dopaminergic degeneration in a primary human dopaminergic neuronal model.
Authors: Cuevas E; Division of Neurotoxicology, National Center for Toxicological Research/US Food and Drug Administration, Jefferson, AR 72079, USA., Guzman A; Division of Neurotoxicology, National Center for Toxicological Research/US Food and Drug Administration, Jefferson, AR 72079, USA., Burks SM; Division of Neurotoxicology, National Center for Toxicological Research/US Food and Drug Administration, Jefferson, AR 72079, USA., Ramirez-Lee A; Division of Neurotoxicology, National Center for Toxicological Research/US Food and Drug Administration, Jefferson, AR 72079, USA., Ali SF; Division of Neurotoxicology, National Center for Toxicological Research/US Food and Drug Administration, Jefferson, AR 72079, USA., Imam SZ; Division of Neurotoxicology, National Center for Toxicological Research/US Food and Drug Administration, Jefferson, AR 72079, USA.
Source: Toxicology reports [Toxicol Rep] 2022 Apr 01; Vol. 9, pp. 806-813. Date of Electronic Publication: 2022 Apr 01 (Print Publication: 2022).
Publication Type: Journal Article
Journal Info: Publisher: Elsevier Ireland Ltd Country of Publication: Ireland NLM ID: 101630272 Publication Model: eCollection Cited Medium: Internet ISSN: 2214-7500 (Electronic) Linking ISSN: 22147500 NLM ISO Abbreviation: Toxicol Rep Subsets: PubMed not MEDLINE
Database: MEDLINE Ultimate
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  Data: Autophagy and protein aggregation as a mechanism of dopaminergic degeneration in a primary human dopaminergic neuronal model.
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  Data: <searchLink fieldCode="AU" term="%22Cuevas+E%22">Cuevas E</searchLink>; Division of Neurotoxicology, National Center for Toxicological Research/US Food and Drug Administration, Jefferson, AR 72079, USA.<br /><searchLink fieldCode="AU" term="%22Guzman+A%22">Guzman A</searchLink>; Division of Neurotoxicology, National Center for Toxicological Research/US Food and Drug Administration, Jefferson, AR 72079, USA.<br /><searchLink fieldCode="AU" term="%22Burks+SM%22">Burks SM</searchLink>; Division of Neurotoxicology, National Center for Toxicological Research/US Food and Drug Administration, Jefferson, AR 72079, USA.<br /><searchLink fieldCode="AU" term="%22Ramirez-Lee+A%22">Ramirez-Lee A</searchLink>; Division of Neurotoxicology, National Center for Toxicological Research/US Food and Drug Administration, Jefferson, AR 72079, USA.<br /><searchLink fieldCode="AU" term="%22Ali+SF%22">Ali SF</searchLink>; Division of Neurotoxicology, National Center for Toxicological Research/US Food and Drug Administration, Jefferson, AR 72079, USA.<br /><searchLink fieldCode="AU" term="%22Imam+SZ%22">Imam SZ</searchLink>; Division of Neurotoxicology, National Center for Toxicological Research/US Food and Drug Administration, Jefferson, AR 72079, USA.
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  Data: <searchLink fieldCode="JN" term="%22101630272%22">Toxicology reports</searchLink> [Toxicol Rep] 2022 Apr 01; Vol. 9, pp. 806-813. <i>Date of Electronic Publication: </i>2022 Apr 01 (<i>Print Publication: </i>2022).
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        Value: 10.1016/j.toxrep.2022.03.047
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              Text: 2022 Apr 01
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