P, P., D, F., H, H., & PA, W. (2023). PLP1-lacZ transgenic mice reveal that splice variants containing "human-specific" exons are relatively minor in comparison to the archetypal transcript and that an upstream regulatory element bolsters expression during early postnatal brain development. Frontiers in cellular neuroscience, 16, 1087145. https://doi.org/10.3389/fncel.2022.1087145
Chicago Style (17th ed.) CitationP, Patyal, Fil D, Hamdan H, and Wight PA. "PLP1-lacZ Transgenic Mice Reveal That Splice Variants Containing "Human-specific" Exons Are Relatively Minor in Comparison to the Archetypal Transcript and That an Upstream Regulatory Element Bolsters Expression During Early Postnatal Brain Development." Frontiers in Cellular Neuroscience 16 (2023): 1087145. https://doi.org/10.3389/fncel.2022.1087145.
MLA (9th ed.) CitationP, Patyal, et al. "PLP1-lacZ Transgenic Mice Reveal That Splice Variants Containing "Human-specific" Exons Are Relatively Minor in Comparison to the Archetypal Transcript and That an Upstream Regulatory Element Bolsters Expression During Early Postnatal Brain Development." Frontiers in Cellular Neuroscience, vol. 16, 2023, p. 1087145, https://doi.org/10.3389/fncel.2022.1087145.