Inflammation and DKK1-induced AKT activation contribute to endothelial dysfunction following NR2F2 loss.
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| Title: | Inflammation and DKK1-induced AKT activation contribute to endothelial dysfunction following NR2F2 loss. |
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| Authors: | Dougherty EJ; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Chen LY; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Awad KS; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Ferreyra GA; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Demirkale CY; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Keshavarz A; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Gairhe S; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Johnston KA; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Hicks ME; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Sandler AB; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Curran CS; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Krack JM; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Ding Y; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Suffredini AF; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Solomon MA; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Elinoff JM; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States., Danner RL; Clinical Center/Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland, United States. |
| Source: | American journal of physiology. Lung cellular and molecular physiology [Am J Physiol Lung Cell Mol Physiol] 2023 Jun 01; Vol. 324 (6), pp. L783-L798. Date of Electronic Publication: 2023 Apr 11. |
| Publication Type: | Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Intramural; Research Support, N.I.H., Extramural |
| Journal Info: | Publisher: American Physiological Society Country of Publication: United States NLM ID: 100901229 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-1504 (Electronic) Linking ISSN: 10400605 NLM ISO Abbreviation: Am J Physiol Lung Cell Mol Physiol Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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