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Loss of TRIM24 promotes IL-10 expression via CBP/p300-dependent IFNβ1 transcription during macrophage activation.

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Title: Loss of TRIM24 promotes IL-10 expression via CBP/p300-dependent IFNβ1 transcription during macrophage activation.
Authors: Hui Z; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China.; Department of Pathogenic Biology and Medical Immunology, School of Basic Medical Sciences, Ningxia Medical University, Yinchuan, 750004, China.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China.; Ningxia Key Laboratory of Prevention and Control of Common Infectious Diseases, Yinchuan, 750004, China., Fu Y; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China., Chen Y; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China., Yin J; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China., Fang H; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China., Tu Y; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China., Gu Y; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China. guyinghz@zju.edu.cn.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China. guyinghz@zju.edu.cn.; Zhejiang Provincial Key Lab of Genetic and Developmental Disorder, Hangzhou, 310058, Zhejiang, China. guyinghz@zju.edu.cn.; Zhejiang Laboratory for Systems & Precision Medicine, Zhejiang University Medical Center, Hangzhou, 311121, China. guyinghz@zju.edu.cn., Zhang J; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China. jwzhang@zju.edu.cn.
Source: Inflammation research : official journal of the European Histamine Research Society ... [et al.] [Inflamm Res] 2023 Jul; Vol. 72 (7), pp. 1441-1452. Date of Electronic Publication: 2023 Jun 16.
Publication Type: Journal Article
Journal Info: Publisher: Birkhäuser Country of Publication: Switzerland NLM ID: 9508160 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1420-908X (Electronic) Linking ISSN: 10233830 NLM ISO Abbreviation: Inflamm Res Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Loss of TRIM24 promotes IL-10 expression via CBP/p300-dependent IFNβ1 transcription during macrophage activation.
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  Data: <searchLink fieldCode="AU" term="%22Hui+Z%22">Hui Z</searchLink>; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China.; Department of Pathogenic Biology and Medical Immunology, School of Basic Medical Sciences, Ningxia Medical University, Yinchuan, 750004, China.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China.; Ningxia Key Laboratory of Prevention and Control of Common Infectious Diseases, Yinchuan, 750004, China.<br /><searchLink fieldCode="AU" term="%22Fu+Y%22">Fu Y</searchLink>; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China.<br /><searchLink fieldCode="AU" term="%22Chen+Y%22">Chen Y</searchLink>; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China.<br /><searchLink fieldCode="AU" term="%22Yin+J%22">Yin J</searchLink>; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China.<br /><searchLink fieldCode="AU" term="%22Fang+H%22">Fang H</searchLink>; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China.<br /><searchLink fieldCode="AU" term="%22Tu+Y%22">Tu Y</searchLink>; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China.<br /><searchLink fieldCode="AU" term="%22Gu+Y%22">Gu Y</searchLink>; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China. guyinghz@zju.edu.cn.; Department of Genetics, Institute of Genetics, School of Medicine, Zhejiang University, Hangzhou, 310058, China. guyinghz@zju.edu.cn.; Zhejiang Provincial Key Lab of Genetic and Developmental Disorder, Hangzhou, 310058, Zhejiang, China. guyinghz@zju.edu.cn.; Zhejiang Laboratory for Systems & Precision Medicine, Zhejiang University Medical Center, Hangzhou, 311121, China. guyinghz@zju.edu.cn.<br /><searchLink fieldCode="AU" term="%22Zhang+J%22">Zhang J</searchLink>; Cancer Institute, Key Laboratory of Cancer Prevention and Intervention, China National Ministry of Education, the Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, 310009, Zhejiang, China. jwzhang@zju.edu.cn.
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        Value: 10.1007/s00011-023-01751-x
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        Text: English
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      – TitleFull: Loss of TRIM24 promotes IL-10 expression via CBP/p300-dependent IFNβ1 transcription during macrophage activation.
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              Text: 2023 Jul
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