APA (7th ed.) Citation

S, S., R, K., G, P., KJ, C., D, B., M, S., . . . V, S. (2023). Deficiency in the neural cell adhesion molecule 2 (NCAM2) reduces axonal levels of beta-site amyloid precursor protein cleaving enzyme 1 (BACE1), affects axonal organization in the hippocampus, and leads to behavioral deficits. Cerebral cortex (New York, N.Y. : 1991), 33(18), 10047. https://doi.org/10.1093/cercor/bhad264

Chicago Style (17th ed.) Citation

S, Sah, Keable R, Pfundstein G, Clemens KJ, Begg D, Schachner M, Leshchyns'ka I, and Sytnyk V. "Deficiency in the Neural Cell Adhesion Molecule 2 (NCAM2) Reduces Axonal Levels of Beta-site Amyloid Precursor Protein Cleaving Enzyme 1 (BACE1), Affects Axonal Organization in the Hippocampus, and Leads to Behavioral Deficits." Cerebral Cortex (New York, N.Y. : 1991) 33, no. 18 (2023): 10047. https://doi.org/10.1093/cercor/bhad264.

MLA (9th ed.) Citation

S, Sah, et al. "Deficiency in the Neural Cell Adhesion Molecule 2 (NCAM2) Reduces Axonal Levels of Beta-site Amyloid Precursor Protein Cleaving Enzyme 1 (BACE1), Affects Axonal Organization in the Hippocampus, and Leads to Behavioral Deficits." Cerebral Cortex (New York, N.Y. : 1991), vol. 33, no. 18, 2023, p. 10047, https://doi.org/10.1093/cercor/bhad264.