Zeb2 drives the formation of CD11c+ atypical B cells to sustain germinal centers that control persistent infection.
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| Title: | Zeb2 drives the formation of CD11c+ atypical B cells to sustain germinal centers that control persistent infection. |
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| Authors: | Gao X; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia., Shen Q; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia.; Francis Crick Institute, London, UK., Roco JA; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia., Dalton B; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia., Frith K; Sydney Children's Hospital, Randwick, Australia.; School of Women's and Children's Health, UNSW Sydney, Sydney, Australia., Munier CML; Kirby Institute, UNSW Sydney, Sydney, Australia., Ballard FD; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia., Wang K; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia., Kelly HG; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia., Nekrasov M; Australian Cancer Research Foundation Biomolecular Resource Facility, John Curtin School of Medical Research, The Australian National University, Canberra, Australia., He JS; ANU Centre for Therapeutic Discovery, John Curtin School of Medical Research, Australian National University, Canberra, Australia., Jaeger R; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia., Carreira P; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia., Ellyard JI; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia., Beattie L; Department of Microbiology and Immunology, University of Melbourne at the Peter Doherty Institute for Infection and Immunity, Melbourne, Australia., Enders A; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia., Cook MC; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia.; Cambridge Institute for Therapeutic Immunology and Infectious Disease, Jeffrey Cheah Biomedical Centre, University of Cambridge, Puddicombe Way, Cambridge CB2 0AW, UK., Zaunders JJ; Centre for Applied Medical Research, St Vincent's Hospital, Sydney, New South Wales, Australia., Cockburn IA; Division of Immunology and Infectious Disease, John Curtin School of Medical Research, The Australian National University, Canberra, Australia. |
| Source: | Science immunology [Sci Immunol] 2024 Mar 29; Vol. 9 (93), pp. eadj4748. Date of Electronic Publication: 2024 Mar 29. |
| Publication Type: | Journal Article; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: American Association for the Advancement of Science Country of Publication: United States NLM ID: 101688624 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2470-9468 (Electronic) Linking ISSN: 24709468 NLM ISO Abbreviation: Sci Immunol Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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