FGF23 facilitates IL-1β synthesis in rheumatoid arthritis through activating PI3K, Akt, and NF-κB pathways.

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Title: FGF23 facilitates IL-1β synthesis in rheumatoid arthritis through activating PI3K, Akt, and NF-κB pathways.
Authors: Hu SL; School of Medicine, China Medical University, Taichung, Taiwan.; Department of Family Medicine, China Medical University Hsinchu Hospital, Hsinchu, Taiwan., Thadevoos LA; Graduate Institute of Biomedical Sciences, China Medical University, Taichung, Taiwan., Ho TL; Graduate Institute of Biomedical Sciences, China Medical University, Taichung, Taiwan., Lin YY; Department of Pharmacology, School of Medicine, China Medical University, Taichung, Taiwan., Chen HT; Department of Sports Medicine, College of Health Care, China Medical University, Taichung, Taiwan.; Department of Orthopedic Surgery, China Medical University Hospital, Taichung, Taiwan., Huang CC; School of Medicine, China Medical University, Taichung, Taiwan.; Division of Immunology and Rheumatology, Department of Internal Medicine, China Medical University Hospital, Taichung, Taiwan., Su CM; Department of Sports Medicine, College of Health Care, China Medical University, Taichung, Taiwan., Tang CH; School of Medicine, China Medical University, Taichung, Taiwan.; Graduate Institute of Biomedical Sciences, China Medical University, Taichung, Taiwan.; Department of Pharmacology, School of Medicine, China Medical University, Taichung, Taiwan.; Chinese Medicine Research Center, China Medical University, Taichung, Taiwan.; Department of Medical Research, China Medical University Hsinchu Hospital, Hsinchu, Taiwan.; Department of Medical Laboratory Science and Biotechnology, College of Medical and Health Science, Asia University, Taichung, Taiwan.
Source: Environmental toxicology [Environ Toxicol] 2024 Jun; Vol. 39 (6), pp. 3283-3291. Date of Electronic Publication: 2024 Feb 21.
Publication Type: Journal Article
Journal Info: Publisher: John Wiley & Sons Country of Publication: United States NLM ID: 100885357 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-7278 (Electronic) Linking ISSN: 15204081 NLM ISO Abbreviation: Environ Toxicol Subsets: MEDLINE
Database: MEDLINE Ultimate
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ISSN:1522-7278
DOI:10.1002/tox.24180