Converting cell death into senescence by PARP1 inhibition improves recovery from acute oxidative injury.
Saved in:
| Title: | Converting cell death into senescence by PARP1 inhibition improves recovery from acute oxidative injury. |
|---|---|
| Authors: | Nehme J; European Research Institute for the Biology of Ageing (ERIBA), University of Groningen (RUG), University Medical Center Groningen, Groningen, the Netherlands.; Department of Biology, Lebanese University, Beirut, Lebanon., Mesilmany L; Department of Biology, Lebanese University, Beirut, Lebanon.; Université de Lille, Inserm, U1003-PHYCEL-Physiologie Cellulaire, Lille, France.; Laboratory of Excellence, Ion Channels Science and Therapeutics, Villeneuve d'Ascq, France., Varela-Eirin M; European Research Institute for the Biology of Ageing (ERIBA), University of Groningen (RUG), University Medical Center Groningen, Groningen, the Netherlands., Brandenburg S; European Research Institute for the Biology of Ageing (ERIBA), University of Groningen (RUG), University Medical Center Groningen, Groningen, the Netherlands., Altulea A; European Research Institute for the Biology of Ageing (ERIBA), University of Groningen (RUG), University Medical Center Groningen, Groningen, the Netherlands., Lin Y; European Research Institute for the Biology of Ageing (ERIBA), University of Groningen (RUG), University Medical Center Groningen, Groningen, the Netherlands., Gaya da Costa M; Department of Internal Medicine, Division of Nephrology, University Medical Center Groningen, University of Groningen, Groningen, the Netherlands., Seelen M; Department of Internal Medicine, Division of Nephrology, University Medical Center Groningen, University of Groningen, Groningen, the Netherlands., Hillebrands JL; Department of Pathology & Medical Biology, Pathology Division, University of Groningen, University Medical Center Groningen, Groningen, the Netherlands., van Goor H; Department of Pathology & Medical Biology, Pathology Division, University of Groningen, University Medical Center Groningen, Groningen, the Netherlands., Saab R; Department of Pediatrics, Stanford University, Stanford, CA, USA., Akl H; Department of Biology, Lebanese University, Beirut, Lebanon.; Department of Natural Sciences, School of Arts and Sciences, Lebanese American University, Beirut, Lebanon., Prevarskaya N; Université de Lille, Inserm, U1003-PHYCEL-Physiologie Cellulaire, Lille, France.; Laboratory of Excellence, Ion Channels Science and Therapeutics, Villeneuve d'Ascq, France., Farfariello V; Université de Lille, Inserm, U1003-PHYCEL-Physiologie Cellulaire, Lille, France.; Laboratory of Excellence, Ion Channels Science and Therapeutics, Villeneuve d'Ascq, France., Demaria M; European Research Institute for the Biology of Ageing (ERIBA), University of Groningen (RUG), University Medical Center Groningen, Groningen, the Netherlands. m.demaria@umcg.nl. |
| Source: | Nature aging [Nat Aging] 2024 Jun; Vol. 4 (6), pp. 771-782. Date of Electronic Publication: 2024 May 09. |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Nature Publishing Group US Country of Publication: United States NLM ID: 101773306 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2662-8465 (Electronic) Linking ISSN: 26628465 NLM ISO Abbreviation: Nat Aging Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
Be the first to leave a comment!
