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CCR2+TREM-1+ monocytes promote natural killer T cell dysfunction contributing towards HBV disease progression.

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Title: CCR2+TREM-1+ monocytes promote natural killer T cell dysfunction contributing towards HBV disease progression.
Authors: Wu X; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China., Zhao W; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China., Miao Q; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China., Shi S; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China., Wei B; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China., Luo L; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China., Cai B; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China. evacaieyou@126.com.
Source: Immunologic research [Immunol Res] 2024 Oct; Vol. 72 (5), pp. 938-947. Date of Electronic Publication: 2024 May 30.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Humana Press Country of Publication: United States NLM ID: 8611087 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1559-0755 (Electronic) Linking ISSN: 0257277X NLM ISO Abbreviation: Immunol Res Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: CCR2+TREM-1+ monocytes promote natural killer T cell dysfunction contributing towards HBV disease progression.
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  Data: <searchLink fieldCode="AU" term="%22Wu+X%22">Wu X</searchLink>; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China.<br /><searchLink fieldCode="AU" term="%22Zhao+W%22">Zhao W</searchLink>; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China.<br /><searchLink fieldCode="AU" term="%22Miao+Q%22">Miao Q</searchLink>; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China.<br /><searchLink fieldCode="AU" term="%22Shi+S%22">Shi S</searchLink>; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China.<br /><searchLink fieldCode="AU" term="%22Wei+B%22">Wei B</searchLink>; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China.<br /><searchLink fieldCode="AU" term="%22Luo+L%22">Luo L</searchLink>; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China.<br /><searchLink fieldCode="AU" term="%22Cai+B%22">Cai B</searchLink>; Department of Laboratory Medicine, Research Center of Clinical Laboratory Medicine, West China Hospital of Sichuan University, Chengdu, Sichuan Province, China. evacaieyou@126.com.
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  Data: <searchLink fieldCode="JN" term="%228611087%22">Immunologic research</searchLink> [Immunol Res] 2024 Oct; Vol. 72 (5), pp. 938-947. <i>Date of Electronic Publication: </i>2024 May 30.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Humana+Press%22">Humana Press </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>8611087 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1559-0755 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%220257277X%22">0257277X </searchLink><i>NLM ISO Abbreviation: </i>Immunol Res <i>Subsets: </i>MEDLINE
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        Value: 10.1007/s12026-024-09495-4
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      – TitleFull: CCR2+TREM-1+ monocytes promote natural killer T cell dysfunction contributing towards HBV disease progression.
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              Text: 2024 Oct
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