APA (7th ed.) Citation

W, Q., M, L., JJ, M., JA, M., S, K., N, H., . . . G, H. (2024). Xenografted human iPSC-derived neurons with the familial Alzheimer's disease APPV717I mutation reveal dysregulated transcriptome signatures linked to synaptic function and implicate LINGO2 as a disease signaling mediator. Acta neuropathologica, 147(1), 107. https://doi.org/10.1007/s00401-024-02755-5

Chicago Style (17th ed.) Citation

W, Qu, et al. "Xenografted Human IPSC-derived Neurons with the Familial Alzheimer's Disease APPV717I Mutation Reveal Dysregulated Transcriptome Signatures Linked to Synaptic Function and Implicate LINGO2 as a Disease Signaling Mediator." Acta Neuropathologica 147, no. 1 (2024): 107. https://doi.org/10.1007/s00401-024-02755-5.

MLA (9th ed.) Citation

W, Qu, et al. "Xenografted Human IPSC-derived Neurons with the Familial Alzheimer's Disease APPV717I Mutation Reveal Dysregulated Transcriptome Signatures Linked to Synaptic Function and Implicate LINGO2 as a Disease Signaling Mediator." Acta Neuropathologica, vol. 147, no. 1, 2024, p. 107, https://doi.org/10.1007/s00401-024-02755-5.