Glyoxal oxidase-mediated detoxification of reactive carbonyl species contributes to virulence, stress tolerance, and development in a pathogenic fungus.

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Title: Glyoxal oxidase-mediated detoxification of reactive carbonyl species contributes to virulence, stress tolerance, and development in a pathogenic fungus.
Authors: Liu X; Genetic Engineering Research Center, School of Life Sciences, Chongqing University, Chongqing, People's Republic of China.; Chongqing Engineering Research Center for Fungal Insecticide, Chongqing, People's Republic of China.; Key Laboratory of Gene Function and Regulation Technologies Under Chongqing Municipal Education Commission, Chongqing, People's Republic of China., Keyhani NO; Department of Biological Sciences, University of Illinois, Chicago, Illinois, United States of America., Liu H; Genetic Engineering Research Center, School of Life Sciences, Chongqing University, Chongqing, People's Republic of China.; Chongqing Engineering Research Center for Fungal Insecticide, Chongqing, People's Republic of China.; Key Laboratory of Gene Function and Regulation Technologies Under Chongqing Municipal Education Commission, Chongqing, People's Republic of China., Zhang Y; Genetic Engineering Research Center, School of Life Sciences, Chongqing University, Chongqing, People's Republic of China.; Chongqing Engineering Research Center for Fungal Insecticide, Chongqing, People's Republic of China.; Key Laboratory of Gene Function and Regulation Technologies Under Chongqing Municipal Education Commission, Chongqing, People's Republic of China., Xia Y; Genetic Engineering Research Center, School of Life Sciences, Chongqing University, Chongqing, People's Republic of China.; Chongqing Engineering Research Center for Fungal Insecticide, Chongqing, People's Republic of China.; Key Laboratory of Gene Function and Regulation Technologies Under Chongqing Municipal Education Commission, Chongqing, People's Republic of China., Cao Y; Genetic Engineering Research Center, School of Life Sciences, Chongqing University, Chongqing, People's Republic of China.; Chongqing Engineering Research Center for Fungal Insecticide, Chongqing, People's Republic of China.; Key Laboratory of Gene Function and Regulation Technologies Under Chongqing Municipal Education Commission, Chongqing, People's Republic of China.
Source: PLoS pathogens [PLoS Pathog] 2024 Jul 30; Vol. 20 (7), pp. e1012431. Date of Electronic Publication: 2024 Jul 30 (Print Publication: 2024).
Publication Type: Journal Article
Journal Info: Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet ISSN: 1553-7374 (Electronic) Linking ISSN: 15537366 NLM ISO Abbreviation: PLoS Pathog Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Glyoxal oxidase-mediated detoxification of reactive carbonyl species contributes to virulence, stress tolerance, and development in a pathogenic fungus.
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  Data: <searchLink fieldCode="AU" term="%22Liu+X%22">Liu X</searchLink>; Genetic Engineering Research Center, School of Life Sciences, Chongqing University, Chongqing, People's Republic of China.; Chongqing Engineering Research Center for Fungal Insecticide, Chongqing, People's Republic of China.; Key Laboratory of Gene Function and Regulation Technologies Under Chongqing Municipal Education Commission, Chongqing, People's Republic of China.<br /><searchLink fieldCode="AU" term="%22Keyhani+NO%22">Keyhani NO</searchLink>; Department of Biological Sciences, University of Illinois, Chicago, Illinois, United States of America.<br /><searchLink fieldCode="AU" term="%22Liu+H%22">Liu H</searchLink>; Genetic Engineering Research Center, School of Life Sciences, Chongqing University, Chongqing, People's Republic of China.; Chongqing Engineering Research Center for Fungal Insecticide, Chongqing, People's Republic of China.; Key Laboratory of Gene Function and Regulation Technologies Under Chongqing Municipal Education Commission, Chongqing, People's Republic of China.<br /><searchLink fieldCode="AU" term="%22Zhang+Y%22">Zhang Y</searchLink>; Genetic Engineering Research Center, School of Life Sciences, Chongqing University, Chongqing, People's Republic of China.; Chongqing Engineering Research Center for Fungal Insecticide, Chongqing, People's Republic of China.; Key Laboratory of Gene Function and Regulation Technologies Under Chongqing Municipal Education Commission, Chongqing, People's Republic of China.<br /><searchLink fieldCode="AU" term="%22Xia+Y%22">Xia Y</searchLink>; Genetic Engineering Research Center, School of Life Sciences, Chongqing University, Chongqing, People's Republic of China.; Chongqing Engineering Research Center for Fungal Insecticide, Chongqing, People's Republic of China.; Key Laboratory of Gene Function and Regulation Technologies Under Chongqing Municipal Education Commission, Chongqing, People's Republic of China.<br /><searchLink fieldCode="AU" term="%22Cao+Y%22">Cao Y</searchLink>; Genetic Engineering Research Center, School of Life Sciences, Chongqing University, Chongqing, People's Republic of China.; Chongqing Engineering Research Center for Fungal Insecticide, Chongqing, People's Republic of China.; Key Laboratory of Gene Function and Regulation Technologies Under Chongqing Municipal Education Commission, Chongqing, People's Republic of China.
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  Data: <searchLink fieldCode="JN" term="%22101238921%22">PLoS pathogens</searchLink> [PLoS Pathog] 2024 Jul 30; Vol. 20 (7), pp. e1012431. <i>Date of Electronic Publication: </i>2024 Jul 30 (<i>Print Publication: </i>2024).
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Public+Library+of+Science%22">Public Library of Science </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>101238921 <i>Publication Model: </i>eCollection <i>Cited Medium: </i>Internet <i>ISSN: </i>1553-7374 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2215537366%22">15537366 </searchLink><i>NLM ISO Abbreviation: </i>PLoS Pathog <i>Subsets: </i>MEDLINE
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        Value: 10.1371/journal.ppat.1012431
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              Text: 2024 Jul 30
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