Tregs delivered post-myocardial infarction adopt an injury-specific phenotype promoting cardiac repair via macrophages in mice.
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| Title: | Tregs delivered post-myocardial infarction adopt an injury-specific phenotype promoting cardiac repair via macrophages in mice. |
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| Authors: | Alshoubaki YK; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia., Nayer B; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia., Lu YZ; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia., Salimova E; Monash Biomedical Imaging, Monash University, Melbourne, Australia., Lau SN; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia., Tan JL; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia., Amann-Zalcenstein D; Advanced Genomics Facility, Advanced Technology and Biology Division, The Walter and Eliza Hall Institute of Medical Research, Parkville, Australia.; Department of Medical Biology, University of Melbourne, Melbourne, Australia., Hickey PF; Advanced Genomics Facility, Advanced Technology and Biology Division, The Walter and Eliza Hall Institute of Medical Research, Parkville, Australia.; Department of Medical Biology, University of Melbourne, Melbourne, Australia., Del Monte-Nieto G; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia.; Victorian Heart Institute, Monash University, Victorian Heart Hospital, Melbourne, Australia., Vasanthakumar A; Olivia Newton-John Cancer Research Institute, Heidelberg, Australia.; La Trobe University, Bundoora, Australia.; Department of Microbiology and Immunology, University of Melbourne, Melbourne, Australia., Martino MM; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia. mikael.martino@monash.edu.; Victorian Heart Institute, Monash University, Victorian Heart Hospital, Melbourne, Australia. mikael.martino@monash.edu. |
| Source: | Nature communications [Nat Commun] 2024 Aug 01; Vol. 15 (1), pp. 6480. Date of Electronic Publication: 2024 Aug 01. |
| Publication Type: | Journal Article; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101528555 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-1723 (Electronic) Linking ISSN: 20411723 NLM ISO Abbreviation: Nat Commun Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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| Header | DbId: mdl DbLabel: MEDLINE Ultimate An: 39090108 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: Tregs delivered post-myocardial infarction adopt an injury-specific phenotype promoting cardiac repair via macrophages in mice. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AU" term="%22Alshoubaki+YK%22">Alshoubaki YK</searchLink>; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia.<br /><searchLink fieldCode="AU" term="%22Nayer+B%22">Nayer B</searchLink>; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia.<br /><searchLink fieldCode="AU" term="%22Lu+YZ%22">Lu YZ</searchLink>; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia.<br /><searchLink fieldCode="AU" term="%22Salimova+E%22">Salimova E</searchLink>; Monash Biomedical Imaging, Monash University, Melbourne, Australia.<br /><searchLink fieldCode="AU" term="%22Lau+SN%22">Lau SN</searchLink>; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia.<br /><searchLink fieldCode="AU" term="%22Tan+JL%22">Tan JL</searchLink>; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia.<br /><searchLink fieldCode="AU" term="%22Amann-Zalcenstein+D%22">Amann-Zalcenstein D</searchLink>; Advanced Genomics Facility, Advanced Technology and Biology Division, The Walter and Eliza Hall Institute of Medical Research, Parkville, Australia.; Department of Medical Biology, University of Melbourne, Melbourne, Australia.<br /><searchLink fieldCode="AU" term="%22Hickey+PF%22">Hickey PF</searchLink>; Advanced Genomics Facility, Advanced Technology and Biology Division, The Walter and Eliza Hall Institute of Medical Research, Parkville, Australia.; Department of Medical Biology, University of Melbourne, Melbourne, Australia.<br /><searchLink fieldCode="AU" term="%22Del+Monte-Nieto+G%22">Del Monte-Nieto G</searchLink>; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia.; Victorian Heart Institute, Monash University, Victorian Heart Hospital, Melbourne, Australia.<br /><searchLink fieldCode="AU" term="%22Vasanthakumar+A%22">Vasanthakumar A</searchLink>; Olivia Newton-John Cancer Research Institute, Heidelberg, Australia.; La Trobe University, Bundoora, Australia.; Department of Microbiology and Immunology, University of Melbourne, Melbourne, Australia.<br /><searchLink fieldCode="AU" term="%22Martino+MM%22">Martino MM</searchLink>; European Molecular Biology Laboratory Australia, Australian Regenerative Medicine Institute, Monash University, Melbourne, Australia. mikael.martino@monash.edu.; Victorian Heart Institute, Monash University, Victorian Heart Hospital, Melbourne, Australia. mikael.martino@monash.edu. – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%22101528555%22">Nature communications</searchLink> [Nat Commun] 2024 Aug 01; Vol. 15 (1), pp. 6480. <i>Date of Electronic Publication: </i>2024 Aug 01. – Name: TypePub Label: Publication Type Group: TypPub Data: Journal Article; Research Support, Non-U.S. Gov't – Name: TitleSource Label: Journal Info Group: Src Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Nature+Pub%2E+Group%22">Nature Pub. Group </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>101528555 <i>Publication Model: </i>Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>2041-1723 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2220411723%22">20411723 </searchLink><i>NLM ISO Abbreviation: </i>Nat Commun <i>Subsets: </i>MEDLINE |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=39090108 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.1038/s41467-024-50806-y Languages: – Code: eng Text: English PhysicalDescription: Pagination: StartPage: 6480 Titles: – TitleFull: Tregs delivered post-myocardial infarction adopt an injury-specific phenotype promoting cardiac repair via macrophages in mice. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Alshoubaki YK – PersonEntity: Name: NameFull: Nayer B – PersonEntity: Name: NameFull: Lu YZ – PersonEntity: Name: NameFull: Salimova E – PersonEntity: Name: NameFull: Lau SN – PersonEntity: Name: NameFull: Tan JL – PersonEntity: Name: NameFull: Amann-Zalcenstein D – PersonEntity: Name: NameFull: Hickey PF – PersonEntity: Name: NameFull: Del Monte-Nieto G – PersonEntity: Name: NameFull: Vasanthakumar A – PersonEntity: Name: NameFull: Martino MM IsPartOfRelationships: – BibEntity: Dates: – D: 01 M: 08 Text: 2024 Aug 01 Type: published Y: 2024 Identifiers: – Type: issn-electronic Value: 2041-1723 Numbering: – Type: volume Value: 15 – Type: issue Value: 1 Titles: – TitleFull: Nature communications Type: main |
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