Enhanced vasoconstriction in sickle cell disease is dependent on ETA receptor activation.

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Title: Enhanced vasoconstriction in sickle cell disease is dependent on ETA receptor activation.
Authors: Allan JM; Section of Cardiorenal Physiology & Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, AL 35233, U.S.A., Fox BM; Section of Cardiorenal Physiology & Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, AL 35233, U.S.A., Kasztan M; Section of Cardiorenal Physiology & Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, AL 35233, U.S.A.; Division of Hematology-Oncology, Department of Pediatrics, University of Alabama at Birmingham, Birmingham, AL 35233, U.S.A., Kelly GC; Section of Cardiorenal Physiology & Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, AL 35233, U.S.A., Molina PA; Section of Cardiorenal Physiology & Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, AL 35233, U.S.A., King MA; Section of Cardiorenal Physiology & Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, AL 35233, U.S.A., Colson J; Section of Cardiorenal Physiology & Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, AL 35233, U.S.A., Wells L; Georgia Prevention Institute, Department of Medicine, Medical College of Georgia at Augusta University, Augusta, GA, U.S.A.; Division of Hematology and Oncology, Department of Medicine, Medical College of Georgia at Augusta University, Augusta, GA, U.S.A., Bowman L; Georgia Prevention Institute, Department of Medicine, Medical College of Georgia at Augusta University, Augusta, GA, U.S.A.; Division of Hematology and Oncology, Department of Medicine, Medical College of Georgia at Augusta University, Augusta, GA, U.S.A., Blackburn M; Georgia Prevention Institute, Department of Medicine, Medical College of Georgia at Augusta University, Augusta, GA, U.S.A.; Division of Hematology and Oncology, Department of Medicine, Medical College of Georgia at Augusta University, Augusta, GA, U.S.A., Kutlar A; Division of Hematology and Oncology, Department of Medicine, Medical College of Georgia at Augusta University, Augusta, GA, U.S.A., Harris RA; Georgia Prevention Institute, Department of Medicine, Medical College of Georgia at Augusta University, Augusta, GA, U.S.A., Pollock DM; Section of Cardiorenal Physiology & Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, AL 35233, U.S.A., Pollock JS; Section of Cardiorenal Physiology & Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Birmingham, AL 35233, U.S.A.
Source: Clinical science (London, England : 1979) [Clin Sci (Lond)] 2024 Dec 04; Vol. 138 (23), pp. 1505-1520.
Publication Type: Journal Article; Randomized Controlled Trial; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Portland Press on behalf of the Medical Research Society and the Biochemical Society Country of Publication: England NLM ID: 7905731 Publication Model: Print Cited Medium: Internet ISSN: 1470-8736 (Electronic) Linking ISSN: 01435221 NLM ISO Abbreviation: Clin Sci (Lond) Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1470-8736
DOI:10.1042/CS20240625