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Apolipoprotein A-I priming via SR-BI and ABCA1 receptor binding upregulates mitochondrial metabolism to promote insulin secretion in INS-1E cells.

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Title: Apolipoprotein A-I priming via SR-BI and ABCA1 receptor binding upregulates mitochondrial metabolism to promote insulin secretion in INS-1E cells.
Authors: Lyons CL; Department of Experimental Medical Sciences, Unit of Medical Protein Science, Lund University, Lund, Sweden., Cowan E; Department of Clinical Sciences, Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Malmö, Sweden., Nilsson O; Department of Experimental Medical Sciences, Unit of Medical Protein Science, Lund University, Lund, Sweden., Mohar M; Department of Experimental Medical Sciences, Unit of Medical Protein Science, Lund University, Lund, Sweden., Peña-Martínez P; Division of Clinical Genetics, Department of Laboratory Medicine, Lund University, Lund, Sweden., Eliasson L; Department of Clinical Sciences, Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Malmö, Sweden., Lagerstedt JO; Department of Experimental Medical Sciences, Unit of Medical Protein Science, Lund University, Lund, Sweden.; Department of Clinical Sciences, Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Malmö, Sweden.
Source: PloS one [PLoS One] 2024 Nov 15; Vol. 19 (11), pp. e0311039. Date of Electronic Publication: 2024 Nov 15 (Print Publication: 2024).
Publication Type: Journal Article
Journal Info: Publisher: Public Library of Science Country of Publication: United States NLM ID: 101285081 Publication Model: eCollection Cited Medium: Internet ISSN: 1932-6203 (Electronic) Linking ISSN: 19326203 NLM ISO Abbreviation: PLoS One Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Apolipoprotein A-I priming via SR-BI and ABCA1 receptor binding upregulates mitochondrial metabolism to promote insulin secretion in INS-1E cells.
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  Data: <searchLink fieldCode="AU" term="%22Lyons+CL%22">Lyons CL</searchLink>; Department of Experimental Medical Sciences, Unit of Medical Protein Science, Lund University, Lund, Sweden.<br /><searchLink fieldCode="AU" term="%22Cowan+E%22">Cowan E</searchLink>; Department of Clinical Sciences, Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Malmö, Sweden.<br /><searchLink fieldCode="AU" term="%22Nilsson+O%22">Nilsson O</searchLink>; Department of Experimental Medical Sciences, Unit of Medical Protein Science, Lund University, Lund, Sweden.<br /><searchLink fieldCode="AU" term="%22Mohar+M%22">Mohar M</searchLink>; Department of Experimental Medical Sciences, Unit of Medical Protein Science, Lund University, Lund, Sweden.<br /><searchLink fieldCode="AU" term="%22Peña-Martínez+P%22">Peña-Martínez P</searchLink>; Division of Clinical Genetics, Department of Laboratory Medicine, Lund University, Lund, Sweden.<br /><searchLink fieldCode="AU" term="%22Eliasson+L%22">Eliasson L</searchLink>; Department of Clinical Sciences, Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Malmö, Sweden.<br /><searchLink fieldCode="AU" term="%22Lagerstedt+JO%22">Lagerstedt JO</searchLink>; Department of Experimental Medical Sciences, Unit of Medical Protein Science, Lund University, Lund, Sweden.; Department of Clinical Sciences, Unit of Islet Cell Exocytosis, Lund University Diabetes Centre, Malmö, Sweden.
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  Data: <searchLink fieldCode="JN" term="%22101285081%22">PloS one</searchLink> [PLoS One] 2024 Nov 15; Vol. 19 (11), pp. e0311039. <i>Date of Electronic Publication: </i>2024 Nov 15 (<i>Print Publication: </i>2024).
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