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EZH2 inhibition sensitizes MYC-high medulloblastoma cancers to PARP inhibition by regulating NUPR1-mediated DNA repair.

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Title: EZH2 inhibition sensitizes MYC-high medulloblastoma cancers to PARP inhibition by regulating NUPR1-mediated DNA repair.
Authors: Yu J; Department of Neurosurgery, Children's Hospital of Fudan University, Shanghai, China., Han J; School of Life Science, Hangzhou Institute for Advanced Study, University of Chinese Academy of Sciences, Hangzhou, China. hanjichang@ucas.ac.cn., Yu M; School of Life Science, Hangzhou Institute for Advanced Study, University of Chinese Academy of Sciences, Hangzhou, China., Rui H; Department of Neurosurgery, Children's Hospital of Fudan University, Shanghai, China., Sun A; Department of Neurosurgery, Huashan Hospital of Fudan University, Shanghai, China. sunan1996@sina.com., Li H; Department of Neurosurgery, Children's Hospital of Fudan University, Shanghai, China. li_hao@fudan.edu.cn.
Source: Oncogene [Oncogene] 2025 Feb; Vol. 44 (6), pp. 391-405. Date of Electronic Publication: 2024 Nov 19.
Publication Type: Journal Article
Journal Info: Publisher: Nature Publishing Group Country of Publication: England NLM ID: 8711562 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1476-5594 (Electronic) Linking ISSN: 09509232 NLM ISO Abbreviation: Oncogene Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: EZH2 inhibition sensitizes MYC-high medulloblastoma cancers to PARP inhibition by regulating NUPR1-mediated DNA repair.
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  Data: <searchLink fieldCode="AU" term="%22Yu+J%22">Yu J</searchLink>; Department of Neurosurgery, Children's Hospital of Fudan University, Shanghai, China.<br /><searchLink fieldCode="AU" term="%22Han+J%22">Han J</searchLink>; School of Life Science, Hangzhou Institute for Advanced Study, University of Chinese Academy of Sciences, Hangzhou, China. hanjichang@ucas.ac.cn.<br /><searchLink fieldCode="AU" term="%22Yu+M%22">Yu M</searchLink>; School of Life Science, Hangzhou Institute for Advanced Study, University of Chinese Academy of Sciences, Hangzhou, China.<br /><searchLink fieldCode="AU" term="%22Rui+H%22">Rui H</searchLink>; Department of Neurosurgery, Children's Hospital of Fudan University, Shanghai, China.<br /><searchLink fieldCode="AU" term="%22Sun+A%22">Sun A</searchLink>; Department of Neurosurgery, Huashan Hospital of Fudan University, Shanghai, China. sunan1996@sina.com.<br /><searchLink fieldCode="AU" term="%22Li+H%22">Li H</searchLink>; Department of Neurosurgery, Children's Hospital of Fudan University, Shanghai, China. li_hao@fudan.edu.cn.
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  Data: <searchLink fieldCode="JN" term="%228711562%22">Oncogene</searchLink> [Oncogene] 2025 Feb; Vol. 44 (6), pp. 391-405. <i>Date of Electronic Publication: </i>2024 Nov 19.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Nature+Publishing+Group%22">Nature Publishing Group </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>8711562 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1476-5594 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2209509232%22">09509232 </searchLink><i>NLM ISO Abbreviation: </i>Oncogene <i>Subsets: </i>MEDLINE
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        Value: 10.1038/s41388-024-03232-9
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              Text: 2025 Feb
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