MLKL deficiency elevates testosterone production in male mice independently of necroptotic functions.
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| Title: | MLKL deficiency elevates testosterone production in male mice independently of necroptotic functions. |
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| Authors: | Chiou S; Walter and Eliza Hall Institute of Medical Research, Parkville, VIC, Australia.; Department of Medical Biology, University of Melbourne, Parkville, VIC, Australia., Cawthorne W; Walter and Eliza Hall Institute of Medical Research, Parkville, VIC, Australia.; Department of Medical Biology, University of Melbourne, Parkville, VIC, Australia., Soerianto T; Department of Biochemistry and Pharmacology, Bio21 Molecular Science and Biotechnology Institute, Parkville, VIC, Australia., Hofferek V; Department of Biochemistry and Pharmacology, Bio21 Molecular Science and Biotechnology Institute, Parkville, VIC, Australia., Patel KM; Walter and Eliza Hall Institute of Medical Research, Parkville, VIC, Australia., Garnish SE; Walter and Eliza Hall Institute of Medical Research, Parkville, VIC, Australia.; Department of Medical Biology, University of Melbourne, Parkville, VIC, Australia., Tovey Crutchfield EC; Walter and Eliza Hall Institute of Medical Research, Parkville, VIC, Australia.; Department of Medical Biology, University of Melbourne, Parkville, VIC, Australia.; Department of Ophthalmology, The Royal Melbourne Hospital, Parkville, VIC, Australia., Hall C; Walter and Eliza Hall Institute of Medical Research, Parkville, VIC, Australia., Hildebrand JM; Walter and Eliza Hall Institute of Medical Research, Parkville, VIC, Australia.; Department of Medical Biology, University of Melbourne, Parkville, VIC, Australia., McConville MJ; Department of Biochemistry and Pharmacology, Bio21 Molecular Science and Biotechnology Institute, Parkville, VIC, Australia., Lawlor KE; Centre for Innate Immunity and Infectious Diseases, Hudson Institute of Medical Research, Clayton, VIC, Australia.; Department of Molecular and Translational Science, Monash University, Clayton, VIC, Australia., Hawkins ED; Walter and Eliza Hall Institute of Medical Research, Parkville, VIC, Australia. hawkins.e@wehi.edu.au.; Department of Medical Biology, University of Melbourne, Parkville, VIC, Australia. hawkins.e@wehi.edu.au., Samson AL; Walter and Eliza Hall Institute of Medical Research, Parkville, VIC, Australia. samson.a@wehi.edu.au.; Department of Medical Biology, University of Melbourne, Parkville, VIC, Australia. samson.a@wehi.edu.au., Murphy JM; Walter and Eliza Hall Institute of Medical Research, Parkville, VIC, Australia. jamesm@wehi.edu.au.; Department of Medical Biology, University of Melbourne, Parkville, VIC, Australia. jamesm@wehi.edu.au.; Drug Discovery Biology, Monash Institute of Pharmaceutical Sciences, Monash University, Parkville, VIC, Australia. jamesm@wehi.edu.au. |
| Source: | Cell death & disease [Cell Death Dis] 2024 Nov 21; Vol. 15 (11), pp. 851. Date of Electronic Publication: 2024 Nov 21. |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101524092 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-4889 (Electronic) NLM ISO Abbreviation: Cell Death Dis Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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