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Amyloid-β-Driven Synaptic Deficits Are Mediated by Synaptic Removal of GluA3-Containing AMPA Receptors.

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Title: Amyloid-β-Driven Synaptic Deficits Are Mediated by Synaptic Removal of GluA3-Containing AMPA Receptors.
Authors: Reinders NR; Netherlands Institute for Neuroscience, Royal Netherlands Academy of Arts and Sciences, Amsterdam 1105 BA, The Netherlands h.w.h.g.kessels@uva.nl n.r.reinders@uu.nl.; Swammerdam Institute of Life Sciences, Amsterdam Neuroscience, University of Amsterdam, Amsterdam 1098 XH, The Netherlands.; Center for Neurogenomics and Cognitive Research, Amsterdam Neuroscience, Vrije Universiteit Amsterdam, Amsterdam 1081 HV, The Netherlands.; Division of Cell Biology, Neurobiology and Biophysics, Department of Biology, Faculty of Science, Utrecht University, Utrecht 3584 CH, The Netherlands., van der Spek SJF; Center for Neurogenomics and Cognitive Research, Amsterdam Neuroscience, Vrije Universiteit Amsterdam, Amsterdam 1081 HV, The Netherlands., Klaassen RV; Center for Neurogenomics and Cognitive Research, Amsterdam Neuroscience, Vrije Universiteit Amsterdam, Amsterdam 1081 HV, The Netherlands., Koymans KJ; Swammerdam Institute of Life Sciences, Amsterdam Neuroscience, University of Amsterdam, Amsterdam 1098 XH, The Netherlands., MacGillavry HD; Division of Cell Biology, Neurobiology and Biophysics, Department of Biology, Faculty of Science, Utrecht University, Utrecht 3584 CH, The Netherlands., Smit AB; Center for Neurogenomics and Cognitive Research, Amsterdam Neuroscience, Vrije Universiteit Amsterdam, Amsterdam 1081 HV, The Netherlands., Kessels HW; Netherlands Institute for Neuroscience, Royal Netherlands Academy of Arts and Sciences, Amsterdam 1105 BA, The Netherlands h.w.h.g.kessels@uva.nl n.r.reinders@uu.nl.; Swammerdam Institute of Life Sciences, Amsterdam Neuroscience, University of Amsterdam, Amsterdam 1098 XH, The Netherlands.
Source: The Journal of neuroscience : the official journal of the Society for Neuroscience [J Neurosci] 2025 Feb 26; Vol. 45 (9). Date of Electronic Publication: 2025 Feb 26.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Society for Neuroscience Country of Publication: United States NLM ID: 8102140 Publication Model: Electronic Cited Medium: Internet ISSN: 1529-2401 (Electronic) Linking ISSN: 02706474 NLM ISO Abbreviation: J Neurosci Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1529-2401
DOI:10.1523/JNEUROSCI.0393-24.2024