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Hypertrophic heart failure promotes gut dysbiosis and gut leakage in interleukin 10-deficient mice.

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Title: Hypertrophic heart failure promotes gut dysbiosis and gut leakage in interleukin 10-deficient mice.
Authors: Ranjan P; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States., Goswami SK; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States., Dutta RK; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States., Kumari R; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States., Colin K; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States.; Division of Clinical Immunology and Rheumatology, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States., Pal HC; Division of Clinical Immunology and Rheumatology, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States., Zhang Q; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States., Lal H; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States., Prasad R; Department of Ophthalmology and Visual Sciences, The University of Alabama at Birmingham, Birmingham, Alabama, United States., Verma SK; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States.; Department of Biomedical Engineering, The University of Alabama at Birmingham, Birmingham, Alabama, United States.
Source: American journal of physiology. Heart and circulatory physiology [Am J Physiol Heart Circ Physiol] 2025 Mar 01; Vol. 328 (3), pp. H447-H459. Date of Electronic Publication: 2025 Jan 24.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
Journal Info: Publisher: American Physiological Society Country of Publication: United States NLM ID: 100901228 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-1539 (Electronic) Linking ISSN: 03636135 NLM ISO Abbreviation: Am J Physiol Heart Circ Physiol Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Hypertrophic heart failure promotes gut dysbiosis and gut leakage in interleukin 10-deficient mice.
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  Data: <searchLink fieldCode="AU" term="%22Ranjan+P%22">Ranjan P</searchLink>; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States.<br /><searchLink fieldCode="AU" term="%22Goswami+SK%22">Goswami SK</searchLink>; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States.<br /><searchLink fieldCode="AU" term="%22Dutta+RK%22">Dutta RK</searchLink>; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States.<br /><searchLink fieldCode="AU" term="%22Kumari+R%22">Kumari R</searchLink>; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States.<br /><searchLink fieldCode="AU" term="%22Colin+K%22">Colin K</searchLink>; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States.; Division of Clinical Immunology and Rheumatology, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States.<br /><searchLink fieldCode="AU" term="%22Pal+HC%22">Pal HC</searchLink>; Division of Clinical Immunology and Rheumatology, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States.<br /><searchLink fieldCode="AU" term="%22Zhang+Q%22">Zhang Q</searchLink>; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States.<br /><searchLink fieldCode="AU" term="%22Lal+H%22">Lal H</searchLink>; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States.<br /><searchLink fieldCode="AU" term="%22Prasad+R%22">Prasad R</searchLink>; Department of Ophthalmology and Visual Sciences, The University of Alabama at Birmingham, Birmingham, Alabama, United States.<br /><searchLink fieldCode="AU" term="%22Verma+SK%22">Verma SK</searchLink>; Division of Cardiovascular Disease, Department of Medicine, The University of Alabama at Birmingham, Birmingham, Alabama, United States.; Department of Biomedical Engineering, The University of Alabama at Birmingham, Birmingham, Alabama, United States.
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  Data: <searchLink fieldCode="JN" term="%22100901228%22">American journal of physiology. Heart and circulatory physiology</searchLink> [Am J Physiol Heart Circ Physiol] 2025 Mar 01; Vol. 328 (3), pp. H447-H459. <i>Date of Electronic Publication: </i>2025 Jan 24.
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  Data: Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22American+Physiological+Society%22">American Physiological Society </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>100901228 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1522-1539 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2203636135%22">03636135 </searchLink><i>NLM ISO Abbreviation: </i>Am J Physiol Heart Circ Physiol <i>Subsets: </i>MEDLINE
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      – Type: doi
        Value: 10.1152/ajpheart.00323.2024
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      – Code: eng
        Text: English
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        StartPage: H447
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            – D: 01
              M: 03
              Text: 2025 Mar 01
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              Y: 2025
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