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Anti-muscarinic 3 antibodies associate with a severe clinical phenotype in patients with systemic sclerosis.

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Title: Anti-muscarinic 3 antibodies associate with a severe clinical phenotype in patients with systemic sclerosis.
Authors: Ayla AY; Division of Rheumatology, University of Texas Health Science Center at Houston, Houston, TX, USA., Kalavar NR; Department of Medicine, University of Texas Health Science Center at Houston, Houston, TX, USA., Pimentel M; Karsh Division of Gastroenterology and MAST Program, Cedars-Sinai Medical Center, Los Angeles, CA, USA., Morales W; Karsh Division of Gastroenterology and MAST Program, Cedars-Sinai Medical Center, Los Angeles, CA, USA., Hummers LK; Division of Rheumatology, Johns Hopkins University School of Medicine, Baltimore, MD, USA., Shah AA; Division of Rheumatology, Johns Hopkins University School of Medicine, Baltimore, MD, USA., Hughes M; Division of Musculoskeletal and Dermatological Sciences, The University of Manchester, Manchester, UK.; Department of Rheumatology, Northern Care Alliance NHS Foundation Trust, Salford Care Organisation, Salford, UK., McMahan ZH; Division of Rheumatology, University of Texas Health Science Center at Houston, Houston, TX, USA.
Source: Rheumatology (Oxford, England) [Rheumatology (Oxford)] 2025 Oct 01; Vol. 64 (10), pp. 5230-5237.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Oxford University Press Country of Publication: England NLM ID: 100883501 Publication Model: Print Cited Medium: Internet ISSN: 1462-0332 (Electronic) Linking ISSN: 14620324 NLM ISO Abbreviation: Rheumatology (Oxford) Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Anti-muscarinic 3 antibodies associate with a severe clinical phenotype in patients with systemic sclerosis.
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  Data: <searchLink fieldCode="AU" term="%22Ayla+AY%22">Ayla AY</searchLink>; Division of Rheumatology, University of Texas Health Science Center at Houston, Houston, TX, USA.<br /><searchLink fieldCode="AU" term="%22Kalavar+NR%22">Kalavar NR</searchLink>; Department of Medicine, University of Texas Health Science Center at Houston, Houston, TX, USA.<br /><searchLink fieldCode="AU" term="%22Pimentel+M%22">Pimentel M</searchLink>; Karsh Division of Gastroenterology and MAST Program, Cedars-Sinai Medical Center, Los Angeles, CA, USA.<br /><searchLink fieldCode="AU" term="%22Morales+W%22">Morales W</searchLink>; Karsh Division of Gastroenterology and MAST Program, Cedars-Sinai Medical Center, Los Angeles, CA, USA.<br /><searchLink fieldCode="AU" term="%22Hummers+LK%22">Hummers LK</searchLink>; Division of Rheumatology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.<br /><searchLink fieldCode="AU" term="%22Shah+AA%22">Shah AA</searchLink>; Division of Rheumatology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.<br /><searchLink fieldCode="AU" term="%22Hughes+M%22">Hughes M</searchLink>; Division of Musculoskeletal and Dermatological Sciences, The University of Manchester, Manchester, UK.; Department of Rheumatology, Northern Care Alliance NHS Foundation Trust, Salford Care Organisation, Salford, UK.<br /><searchLink fieldCode="AU" term="%22McMahan+ZH%22">McMahan ZH</searchLink>; Division of Rheumatology, University of Texas Health Science Center at Houston, Houston, TX, USA.
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  Data: <searchLink fieldCode="JN" term="%22100883501%22">Rheumatology (Oxford, England)</searchLink> [Rheumatology (Oxford)] 2025 Oct 01; Vol. 64 (10), pp. 5230-5237.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Oxford+University+Press%22">Oxford University Press </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>100883501 <i>Publication Model: </i>Print <i>Cited Medium: </i>Internet <i>ISSN: </i>1462-0332 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2214620324%22">14620324 </searchLink><i>NLM ISO Abbreviation: </i>Rheumatology (Oxford) <i>Subsets: </i>MEDLINE
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        Value: 10.1093/rheumatology/keaf197
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      – TitleFull: Anti-muscarinic 3 antibodies associate with a severe clinical phenotype in patients with systemic sclerosis.
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              Text: 2025 Oct 01
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              Y: 2025
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