APA (7th ed.) Citation

K, V., S, G., R, V., A, N., R, L., S, C., . . . Jr, L. N. (2025). In vivo CRISPR screening identifies NF1/RASA1/TP53 co-mutations and downstream MEK signaling as a common key mechanism of sinonasal tumorigenesis. BioRxiv : the preprint server for biology. https://doi.org/10.1101/2025.05.19.654661

Chicago Style (17th ed.) Citation

K, Vu, et al. "In Vivo CRISPR Screening Identifies NF1/RASA1/TP53 Co-mutations and Downstream MEK Signaling as a Common Key Mechanism of Sinonasal Tumorigenesis." BioRxiv : The Preprint Server for Biology 2025. https://doi.org/10.1101/2025.05.19.654661.

MLA (9th ed.) Citation

K, Vu, et al. "In Vivo CRISPR Screening Identifies NF1/RASA1/TP53 Co-mutations and Downstream MEK Signaling as a Common Key Mechanism of Sinonasal Tumorigenesis." BioRxiv : The Preprint Server for Biology, 2025, https://doi.org/10.1101/2025.05.19.654661.