Luteolin delays the progression of IgA nephropathy by attenuating inflammation, oxidative stress and reducing extracellular matrix accumulation through activating the Nrf-2/HO-1 pathway.

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Title: Luteolin delays the progression of IgA nephropathy by attenuating inflammation, oxidative stress and reducing extracellular matrix accumulation through activating the Nrf-2/HO-1 pathway.
Authors: Liang DY; Clinical Research Center, Jiading District Central Hospital Affiliated Shanghai University of Medicine and Health Sciences, Shanghai, China., Cong SH; Department of Stomatology, Jiading District Central Hospital Affiliated Shanghai University of Medicine and Health Sciences, Shanghai, China., Li LH; Department of Scientific Research, Jiading District Central Hospital Affiliated Shanghai University of Medicine and Health Sciences, Shanghai, China., Yi QQ; Clinical Research Center, Jiading District Central Hospital Affiliated Shanghai University of Medicine and Health Sciences, Shanghai, China., Tang LP; Clinical Research Center, Jiading District Central Hospital Affiliated Shanghai University of Medicine and Health Sciences, Shanghai, China., Cao LO; Department of Nephrology, Molecular Cell Lab for Kidney Disease, Ren Ji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Source: Frontiers in pharmacology [Front Pharmacol] 2025 Jul 08; Vol. 16, pp. 1530655. Date of Electronic Publication: 2025 Jul 08 (Print Publication: 2025).
Publication Type: Journal Article
Journal Info: Publisher: Frontiers Media] Country of Publication: Switzerland NLM ID: 101548923 Publication Model: eCollection Cited Medium: Print ISSN: 1663-9812 (Print) Linking ISSN: 16639812 NLM ISO Abbreviation: Front Pharmacol Subsets: PubMed not MEDLINE
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  Data: Luteolin delays the progression of IgA nephropathy by attenuating inflammation, oxidative stress and reducing extracellular matrix accumulation through activating the Nrf-2/HO-1 pathway.
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  Data: <searchLink fieldCode="AU" term="%22Liang+DY%22">Liang DY</searchLink>; Clinical Research Center, Jiading District Central Hospital Affiliated Shanghai University of Medicine and Health Sciences, Shanghai, China.<br /><searchLink fieldCode="AU" term="%22Cong+SH%22">Cong SH</searchLink>; Department of Stomatology, Jiading District Central Hospital Affiliated Shanghai University of Medicine and Health Sciences, Shanghai, China.<br /><searchLink fieldCode="AU" term="%22Li+LH%22">Li LH</searchLink>; Department of Scientific Research, Jiading District Central Hospital Affiliated Shanghai University of Medicine and Health Sciences, Shanghai, China.<br /><searchLink fieldCode="AU" term="%22Yi+QQ%22">Yi QQ</searchLink>; Clinical Research Center, Jiading District Central Hospital Affiliated Shanghai University of Medicine and Health Sciences, Shanghai, China.<br /><searchLink fieldCode="AU" term="%22Tang+LP%22">Tang LP</searchLink>; Clinical Research Center, Jiading District Central Hospital Affiliated Shanghai University of Medicine and Health Sciences, Shanghai, China.<br /><searchLink fieldCode="AU" term="%22Cao+LO%22">Cao LO</searchLink>; Department of Nephrology, Molecular Cell Lab for Kidney Disease, Ren Ji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
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  Data: <searchLink fieldCode="JN" term="%22101548923%22">Frontiers in pharmacology</searchLink> [Front Pharmacol] 2025 Jul 08; Vol. 16, pp. 1530655. <i>Date of Electronic Publication: </i>2025 Jul 08 (<i>Print Publication: </i>2025).
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Frontiers+Media]%22">Frontiers Media] </searchLink><i>Country of Publication: </i>Switzerland <i>NLM ID: </i>101548923 <i>Publication Model: </i>eCollection <i>Cited Medium: </i>Print <i>ISSN: </i>1663-9812 (Print) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2216639812%22">16639812 </searchLink><i>NLM ISO Abbreviation: </i>Front Pharmacol <i>Subsets: </i>PubMed not MEDLINE
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        Value: 10.3389/fphar.2025.1530655
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      – TitleFull: Luteolin delays the progression of IgA nephropathy by attenuating inflammation, oxidative stress and reducing extracellular matrix accumulation through activating the Nrf-2/HO-1 pathway.
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              Text: 2025 Jul 08
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