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Liver CYP4A autophagic lysosomal degradation: A major role for the autophagic receptor SQSTM1/p62 through an uncommon target interaction site.

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Title: Liver CYP4A autophagic lysosomal degradation: A major role for the autophagic receptor SQSTM1/p62 through an uncommon target interaction site.
Authors: He L; Department of Cellular & Molecular Pharmacology, University of California San Francisco, San Francisco, California., Kwon D; Department of Cellular & Molecular Pharmacology, University of California San Francisco, San Francisco, California., Trnka MJ; Department of Pharmaceutical Chemistry, University of California San Francisco, San Francisco, California., Liu Y; Department of Cellular & Molecular Pharmacology, University of California San Francisco, San Francisco, California., Yang J; Department of Medicinal Chemistry, University of Washington, Seattle, Washington., Li KH; Department of Pharmaceutical Chemistry, University of California San Francisco, San Francisco, California., Totah RA; Department of Medicinal Chemistry, University of Washington, Seattle, Washington., Johnson EF; Department of Molecular Medicine, Scripps Research Institute, La Jolla, California., Burlingame AL; Department of Pharmaceutical Chemistry, University of California San Francisco, San Francisco, California., Correia MA; Department of Cellular & Molecular Pharmacology, University of California San Francisco, San Francisco, California; Department of Pharmaceutical Chemistry, University of California San Francisco, San Francisco, California; Department of Bioengineering and Therapeutic Sciences, University of California San Francisco, San Francisco, California; The Liver Center, University of California San Francisco, San Francisco, California. Electronic address: almira.correia@ucsf.edu.
Source: Molecular pharmacology [Mol Pharmacol] 2025 Sep; Vol. 107 (9), pp. 100061. Date of Electronic Publication: 2025 Jul 11.
Publication Type: Journal Article
Journal Info: Publisher: Elsevier Country of Publication: Netherlands NLM ID: 0035623 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1521-0111 (Electronic) Linking ISSN: 0026895X NLM ISO Abbreviation: Mol Pharmacol Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: <searchLink fieldCode="AU" term="%22He+L%22">He L</searchLink>; Department of Cellular & Molecular Pharmacology, University of California San Francisco, San Francisco, California.<br /><searchLink fieldCode="AU" term="%22Kwon+D%22">Kwon D</searchLink>; Department of Cellular & Molecular Pharmacology, University of California San Francisco, San Francisco, California.<br /><searchLink fieldCode="AU" term="%22Trnka+MJ%22">Trnka MJ</searchLink>; Department of Pharmaceutical Chemistry, University of California San Francisco, San Francisco, California.<br /><searchLink fieldCode="AU" term="%22Liu+Y%22">Liu Y</searchLink>; Department of Cellular & Molecular Pharmacology, University of California San Francisco, San Francisco, California.<br /><searchLink fieldCode="AU" term="%22Yang+J%22">Yang J</searchLink>; Department of Medicinal Chemistry, University of Washington, Seattle, Washington.<br /><searchLink fieldCode="AU" term="%22Li+KH%22">Li KH</searchLink>; Department of Pharmaceutical Chemistry, University of California San Francisco, San Francisco, California.<br /><searchLink fieldCode="AU" term="%22Totah+RA%22">Totah RA</searchLink>; Department of Medicinal Chemistry, University of Washington, Seattle, Washington.<br /><searchLink fieldCode="AU" term="%22Johnson+EF%22">Johnson EF</searchLink>; Department of Molecular Medicine, Scripps Research Institute, La Jolla, California.<br /><searchLink fieldCode="AU" term="%22Burlingame+AL%22">Burlingame AL</searchLink>; Department of Pharmaceutical Chemistry, University of California San Francisco, San Francisco, California.<br /><searchLink fieldCode="AU" term="%22Correia+MA%22">Correia MA</searchLink>; Department of Cellular & Molecular Pharmacology, University of California San Francisco, San Francisco, California; Department of Pharmaceutical Chemistry, University of California San Francisco, San Francisco, California; Department of Bioengineering and Therapeutic Sciences, University of California San Francisco, San Francisco, California; The Liver Center, University of California San Francisco, San Francisco, California. Electronic address: almira.correia@ucsf.edu.
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              Text: 2025 Sep
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