ACAA1 knockout increases the survival rate of KPC mice by activating autophagy.
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| Title: | ACAA1 knockout increases the survival rate of KPC mice by activating autophagy. |
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| Authors: | Lee H; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea., Kang M; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea., Sim SH; Division of Rare and Refractory Cancer, Research Institute, National Cancer Center, Goyang, Republic of Korea., Kang JH; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea., Choi W; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea; Division of Rare and Refractory Cancer, Research Institute, National Cancer Center, Goyang, Republic of Korea., Chun JW; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea; Division of Clinical Research, Research Institute, National Cancer Center, Goyang, Republic of Korea; Center for Liver and Pancreatobiliary Cancer, National Cancer Center, Goyang, Republic of Korea., Hong W; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea., Kim C; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea., Ham W; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea., Park JH; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea., Koh EB; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea., Jeon Y; Laboratory Animal Research Facility, Research Institute, National Cancer Center, Goyang, Republic of Korea., Woo SM; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea; Division of Rare and Refractory Cancer, Research Institute, National Cancer Center, Goyang, Republic of Korea; Center for Liver and Pancreatobiliary Cancer, National Cancer Center, Goyang, Republic of Korea., Kim SY; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea. Electronic address: tgase@ncc.re.kr. |
| Source: | Molecular metabolism [Mol Metab] 2025 Oct; Vol. 100, pp. 102237. Date of Electronic Publication: 2025 Aug 21. |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Elsevier GmbH Country of Publication: Germany NLM ID: 101605730 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2212-8778 (Electronic) Linking ISSN: 22128778 NLM ISO Abbreviation: Mol Metab Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
| FullText | Text: Availability: 0 |
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| Header | DbId: mdl DbLabel: MEDLINE Ultimate An: 40848971 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: ACAA1 knockout increases the survival rate of KPC mice by activating autophagy. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AU" term="%22Lee+H%22">Lee H</searchLink>; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Kang+M%22">Kang M</searchLink>; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Sim+SH%22">Sim SH</searchLink>; Division of Rare and Refractory Cancer, Research Institute, National Cancer Center, Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Kang+JH%22">Kang JH</searchLink>; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Choi+W%22">Choi W</searchLink>; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea; Division of Rare and Refractory Cancer, Research Institute, National Cancer Center, Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Chun+JW%22">Chun JW</searchLink>; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea; Division of Clinical Research, Research Institute, National Cancer Center, Goyang, Republic of Korea; Center for Liver and Pancreatobiliary Cancer, National Cancer Center, Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Hong+W%22">Hong W</searchLink>; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Kim+C%22">Kim C</searchLink>; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Ham+W%22">Ham W</searchLink>; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Park+JH%22">Park JH</searchLink>; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Koh+EB%22">Koh EB</searchLink>; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Jeon+Y%22">Jeon Y</searchLink>; Laboratory Animal Research Facility, Research Institute, National Cancer Center, Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Woo+SM%22">Woo SM</searchLink>; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea; Division of Rare and Refractory Cancer, Research Institute, National Cancer Center, Goyang, Republic of Korea; Center for Liver and Pancreatobiliary Cancer, National Cancer Center, Goyang, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Kim+SY%22">Kim SY</searchLink>; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea. Electronic address: tgase@ncc.re.kr. – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%22101605730%22">Molecular metabolism</searchLink> [Mol Metab] 2025 Oct; Vol. 100, pp. 102237. <i>Date of Electronic Publication: </i>2025 Aug 21. – Name: TypePub Label: Publication Type Group: TypPub Data: Journal Article – Name: TitleSource Label: Journal Info Group: Src Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Elsevier+GmbH%22">Elsevier GmbH </searchLink><i>Country of Publication: </i>Germany <i>NLM ID: </i>101605730 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>2212-8778 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2222128778%22">22128778 </searchLink><i>NLM ISO Abbreviation: </i>Mol Metab <i>Subsets: </i>MEDLINE |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=40848971 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.1016/j.molmet.2025.102237 Languages: – Code: eng Text: English PhysicalDescription: Pagination: StartPage: 102237 Titles: – TitleFull: ACAA1 knockout increases the survival rate of KPC mice by activating autophagy. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Lee H – PersonEntity: Name: NameFull: Kang M – PersonEntity: Name: NameFull: Sim SH – PersonEntity: Name: NameFull: Kang JH – PersonEntity: Name: NameFull: Choi W – PersonEntity: Name: NameFull: Chun JW – PersonEntity: Name: NameFull: Hong W – PersonEntity: Name: NameFull: Kim C – PersonEntity: Name: NameFull: Ham W – PersonEntity: Name: NameFull: Park JH – PersonEntity: Name: NameFull: Koh EB – PersonEntity: Name: NameFull: Jeon Y – PersonEntity: Name: NameFull: Woo SM – PersonEntity: Name: NameFull: Kim SY IsPartOfRelationships: – BibEntity: Dates: – D: 01 M: 10 Text: 2025 Oct Type: published Y: 2025 Identifiers: – Type: issn-electronic Value: 2212-8778 Numbering: – Type: volume Value: 100 Titles: – TitleFull: Molecular metabolism Type: main |
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