ACAA1 knockout increases the survival rate of KPC mice by activating autophagy.
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| Title: | ACAA1 knockout increases the survival rate of KPC mice by activating autophagy. |
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| Authors: | Lee H; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea., Kang M; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea., Sim SH; Division of Rare and Refractory Cancer, Research Institute, National Cancer Center, Goyang, Republic of Korea., Kang JH; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea., Choi W; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea; Division of Rare and Refractory Cancer, Research Institute, National Cancer Center, Goyang, Republic of Korea., Chun JW; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea; Division of Clinical Research, Research Institute, National Cancer Center, Goyang, Republic of Korea; Center for Liver and Pancreatobiliary Cancer, National Cancer Center, Goyang, Republic of Korea., Hong W; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea., Kim C; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea., Ham W; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea., Park JH; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea., Koh EB; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea., Jeon Y; Laboratory Animal Research Facility, Research Institute, National Cancer Center, Goyang, Republic of Korea., Woo SM; Department of Cancer Biomedical Science, Graduate School of Cancer Science and Policy, National Cancer Center, Goyang, Republic of Korea; Division of Rare and Refractory Cancer, Research Institute, National Cancer Center, Goyang, Republic of Korea; Center for Liver and Pancreatobiliary Cancer, National Cancer Center, Goyang, Republic of Korea., Kim SY; Division of Cancer Biology, Research Institute, National Cancer Center, Goyang, Republic of Korea; New Cancer Cure-Bio Co., Goyang, Republic of Korea. Electronic address: tgase@ncc.re.kr. |
| Source: | Molecular metabolism [Mol Metab] 2025 Oct; Vol. 100, pp. 102237. Date of Electronic Publication: 2025 Aug 21. |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Elsevier GmbH Country of Publication: Germany NLM ID: 101605730 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2212-8778 (Electronic) Linking ISSN: 22128778 NLM ISO Abbreviation: Mol Metab Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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