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Garadacimab in hereditary angioedema due to normal C1INH with F12/PLG mutations.

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Title: Garadacimab in hereditary angioedema due to normal C1INH with F12/PLG mutations.
Authors: Cohn DM; Department of Vascular Medicine, Amsterdam Cardiovascular Sciences, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands. Electronic address: d.m.cohn@amsterdamumc.nl., Reshef A; Allergy, Immunology, and Angioedema Center, Barzilai University Hospital, Ashkelon, Israel., Staubach P; Department of Dermatology, University Medical Center Mainz, Johannes Gutenberg University, Mainz, Germany., Lumry WR; AARA Research Center, Dallas, Tex., Feuersenger H; CSL Innovation GmbH, Marburg, Germany., Jacobs I; CSL Behring, King of Prussia, Pa., Pragst I; CSL Innovation GmbH, Marburg, Germany., Zuraw B; Division of Allergy and Immunology, Department of Medicine, University of California San Diego, La Jolla, Calif; San Diego VA Healthcare, La Jolla, Calif., Bork K; Department of Dermatology, University Medical Center Mainz, Johannes Gutenberg University, Mainz, Germany.
Source: The Journal of allergy and clinical immunology [J Allergy Clin Immunol] 2025 Dec; Vol. 156 (6), pp. 1756-1760.e4. Date of Electronic Publication: 2025 Aug 26.
Publication Type: Journal Article; Clinical Trial, Phase II; Clinical Trial, Phase III; Multicenter Study
Journal Info: Publisher: Mosby Country of Publication: United States NLM ID: 1275002 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1097-6825 (Electronic) Linking ISSN: 00916749 NLM ISO Abbreviation: J Allergy Clin Immunol Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Garadacimab in hereditary angioedema due to normal C1INH with F12/PLG mutations.
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  Data: <searchLink fieldCode="AU" term="%22Cohn+DM%22">Cohn DM</searchLink>; Department of Vascular Medicine, Amsterdam Cardiovascular Sciences, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands. Electronic address: d.m.cohn@amsterdamumc.nl.<br /><searchLink fieldCode="AU" term="%22Reshef+A%22">Reshef A</searchLink>; Allergy, Immunology, and Angioedema Center, Barzilai University Hospital, Ashkelon, Israel.<br /><searchLink fieldCode="AU" term="%22Staubach+P%22">Staubach P</searchLink>; Department of Dermatology, University Medical Center Mainz, Johannes Gutenberg University, Mainz, Germany.<br /><searchLink fieldCode="AU" term="%22Lumry+WR%22">Lumry WR</searchLink>; AARA Research Center, Dallas, Tex.<br /><searchLink fieldCode="AU" term="%22Feuersenger+H%22">Feuersenger H</searchLink>; CSL Innovation GmbH, Marburg, Germany.<br /><searchLink fieldCode="AU" term="%22Jacobs+I%22">Jacobs I</searchLink>; CSL Behring, King of Prussia, Pa.<br /><searchLink fieldCode="AU" term="%22Pragst+I%22">Pragst I</searchLink>; CSL Innovation GmbH, Marburg, Germany.<br /><searchLink fieldCode="AU" term="%22Zuraw+B%22">Zuraw B</searchLink>; Division of Allergy and Immunology, Department of Medicine, University of California San Diego, La Jolla, Calif; San Diego VA Healthcare, La Jolla, Calif.<br /><searchLink fieldCode="AU" term="%22Bork+K%22">Bork K</searchLink>; Department of Dermatology, University Medical Center Mainz, Johannes Gutenberg University, Mainz, Germany.
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  Data: <searchLink fieldCode="JN" term="%221275002%22">The Journal of allergy and clinical immunology</searchLink> [J Allergy Clin Immunol] 2025 Dec; Vol. 156 (6), pp. 1756-1760.e4. <i>Date of Electronic Publication: </i>2025 Aug 26.
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  Data: Journal Article; Clinical Trial, Phase II; Clinical Trial, Phase III; Multicenter Study
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Mosby%22">Mosby </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>1275002 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1097-6825 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2200916749%22">00916749 </searchLink><i>NLM ISO Abbreviation: </i>J Allergy Clin Immunol <i>Subsets: </i>MEDLINE
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        Value: 10.1016/j.jaci.2025.08.005
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        Text: English
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      – TitleFull: Garadacimab in hereditary angioedema due to normal C1INH with F12/PLG mutations.
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              M: 12
              Text: 2025 Dec
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              Y: 2025
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