Targeting mitochondrial proteases CLPP and LONP1 via disruption of mitochondrial redox homeostasis induces proteotoxic stress and suppresses tumor progression.
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| Title: | Targeting mitochondrial proteases CLPP and LONP1 via disruption of mitochondrial redox homeostasis induces proteotoxic stress and suppresses tumor progression. |
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| Authors: | Nandha SR; Radiation Biology & Health Sciences Division, Bio-science Group, Bhabha Atomic Research Centre, Trombay, Mumbai, 400 085, India.; Homi Bhabha National Institute, Anushaktinagar, Mumbai, 400 094, India., Patwardhan RS; Radiation Biology & Health Sciences Division, Bio-science Group, Bhabha Atomic Research Centre, Trombay, Mumbai, 400 085, India.; Homi Bhabha National Institute, Anushaktinagar, Mumbai, 400 094, India., Sharma D; Radiation Biology & Health Sciences Division, Bio-science Group, Bhabha Atomic Research Centre, Trombay, Mumbai, 400 085, India.; Homi Bhabha National Institute, Anushaktinagar, Mumbai, 400 094, India., Sandur SK; Radiation Biology & Health Sciences Division, Bio-science Group, Bhabha Atomic Research Centre, Trombay, Mumbai, 400 085, India. sskumar@barc.gov.in.; Homi Bhabha National Institute, Anushaktinagar, Mumbai, 400 094, India. sskumar@barc.gov.in., Checker R; Radiation Biology & Health Sciences Division, Bio-science Group, Bhabha Atomic Research Centre, Trombay, Mumbai, 400 085, India. rchecker@barc.gov.in.; Homi Bhabha National Institute, Anushaktinagar, Mumbai, 400 094, India. rchecker@barc.gov.in. |
| Source: | Cell communication and signaling : CCS [Cell Commun Signal] 2025 Sep 03; Vol. 23 (1), pp. 393. Date of Electronic Publication: 2025 Sep 03. |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: BioMed Central Country of Publication: England NLM ID: 101170464 Publication Model: Electronic Cited Medium: Internet ISSN: 1478-811X (Electronic) Linking ISSN: 1478811X NLM ISO Abbreviation: Cell Commun Signal Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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| ISSN: | 1478-811X |
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| DOI: | 10.1186/s12964-025-02127-w |
