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Pancreatic Cells Are Resistant to KRASQ61L Expression due to Hyperactive ERK/MAPK Signaling and Apoptosis Induction.

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Title: Pancreatic Cells Are Resistant to KRASQ61L Expression due to Hyperactive ERK/MAPK Signaling and Apoptosis Induction.
Authors: Burge RA; Department of Biochemistry & Molecular Biology, Medical University of South Carolina, Charleston, South Carolina., Bialousow L; Department of Biochemistry & Molecular Biology, Medical University of South Carolina, Charleston, South Carolina., McFall T; Department of Biochemistry and MCW Cancer Center, Medical College of Wisconsin, Milwaukee, Wisconsin., Bamonte L; Department of Biochemistry & Molecular Biology, Medical University of South Carolina, Charleston, South Carolina., Johnson G; Department of Biochemistry & Molecular Biology, Medical University of South Carolina, Charleston, South Carolina., Smith M; College of Graduate Studies, Medical University of South Carolina, Charleston, South Carolina., Vaena SG; Bioinformatics Core, Medical University of South Carolina, Charleston, South Carolina., Comte-Walters S; Department of Pharmacology and Immunology, Medical University of South Carolina, Charleston, South Carolina., Ball LE; Department of Pharmacology and Immunology, Medical University of South Carolina, Charleston, South Carolina.; Hollings Cancer Center, Medical University of South Carolina, Charleston, South Carolina., Berto S; Bioinformatics Core, Medical University of South Carolina, Charleston, South Carolina.; Department of Neuroscience, Medical University of South Carolina, Charleston, South Carolina.; Hollings Cancer Center, Medical University of South Carolina, Charleston, South Carolina., O'Bryan JP; Department of Biochemistry & Molecular Biology, Medical University of South Carolina, Charleston, South Carolina.; Hollings Cancer Center, Medical University of South Carolina, Charleston, South Carolina., Hobbs GA; Department of Biochemistry & Molecular Biology, Medical University of South Carolina, Charleston, South Carolina.; Hollings Cancer Center, Medical University of South Carolina, Charleston, South Carolina.
Source: Cancer research communications [Cancer Res Commun] 2025 Oct 01; Vol. 5 (10), pp. 1865-1878.
Publication Type: Journal Article
Journal Info: Publisher: American Association for Cancer Research Country of Publication: United States NLM ID: 9918281580506676 Publication Model: Print Cited Medium: Internet ISSN: 2767-9764 (Electronic) Linking ISSN: 27679764 NLM ISO Abbreviation: Cancer Res Commun Subsets: MEDLINE
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  Data: Pancreatic Cells Are Resistant to KRASQ61L Expression due to Hyperactive ERK/MAPK Signaling and Apoptosis Induction.
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  Data: <searchLink fieldCode="AU" term="%22Burge+RA%22">Burge RA</searchLink>; Department of Biochemistry & Molecular Biology, Medical University of South Carolina, Charleston, South Carolina.<br /><searchLink fieldCode="AU" term="%22Bialousow+L%22">Bialousow L</searchLink>; Department of Biochemistry & Molecular Biology, Medical University of South Carolina, Charleston, South Carolina.<br /><searchLink fieldCode="AU" term="%22McFall+T%22">McFall T</searchLink>; Department of Biochemistry and MCW Cancer Center, Medical College of Wisconsin, Milwaukee, Wisconsin.<br /><searchLink fieldCode="AU" term="%22Bamonte+L%22">Bamonte L</searchLink>; Department of Biochemistry & Molecular Biology, Medical University of South Carolina, Charleston, South Carolina.<br /><searchLink fieldCode="AU" term="%22Johnson+G%22">Johnson G</searchLink>; Department of Biochemistry & Molecular Biology, Medical University of South Carolina, Charleston, South Carolina.<br /><searchLink fieldCode="AU" term="%22Smith+M%22">Smith M</searchLink>; College of Graduate Studies, Medical University of South Carolina, Charleston, South Carolina.<br /><searchLink fieldCode="AU" term="%22Vaena+SG%22">Vaena SG</searchLink>; Bioinformatics Core, Medical University of South Carolina, Charleston, South Carolina.<br /><searchLink fieldCode="AU" term="%22Comte-Walters+S%22">Comte-Walters S</searchLink>; Department of Pharmacology and Immunology, Medical University of South Carolina, Charleston, South Carolina.<br /><searchLink fieldCode="AU" term="%22Ball+LE%22">Ball LE</searchLink>; Department of Pharmacology and Immunology, Medical University of South Carolina, Charleston, South Carolina.; Hollings Cancer Center, Medical University of South Carolina, Charleston, South Carolina.<br /><searchLink fieldCode="AU" term="%22Berto+S%22">Berto S</searchLink>; Bioinformatics Core, Medical University of South Carolina, Charleston, South Carolina.; Department of Neuroscience, Medical University of South Carolina, Charleston, South Carolina.; Hollings Cancer Center, Medical University of South Carolina, Charleston, South Carolina.<br /><searchLink fieldCode="AU" term="%22O'Bryan+JP%22">O'Bryan JP</searchLink>; Department of Biochemistry & Molecular Biology, Medical University of South Carolina, Charleston, South Carolina.; Hollings Cancer Center, Medical University of South Carolina, Charleston, South Carolina.<br /><searchLink fieldCode="AU" term="%22Hobbs+GA%22">Hobbs GA</searchLink>; Department of Biochemistry & Molecular Biology, Medical University of South Carolina, Charleston, South Carolina.; Hollings Cancer Center, Medical University of South Carolina, Charleston, South Carolina.
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  Data: <searchLink fieldCode="JN" term="%229918281580506676%22">Cancer research communications</searchLink> [Cancer Res Commun] 2025 Oct 01; Vol. 5 (10), pp. 1865-1878.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22American+Association+for+Cancer+Research%22">American Association for Cancer Research </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>9918281580506676 <i>Publication Model: </i>Print <i>Cited Medium: </i>Internet <i>ISSN: </i>2767-9764 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2227679764%22">27679764 </searchLink><i>NLM ISO Abbreviation: </i>Cancer Res Commun <i>Subsets: </i>MEDLINE
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