APA (7th ed.) Citation

M, K., GA, H., R, D., D, D., J, H., JF, L., . . . T, M. (2026). Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma. Cancer research, 86(8), 2042. https://doi.org/10.1158/0008-5472.CAN-25-0018

Chicago Style (17th ed.) Citation

M, Kamgar, et al. "Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma." Cancer Research 86, no. 8 (2026): 2042. https://doi.org/10.1158/0008-5472.CAN-25-0018.

MLA (9th ed.) Citation

M, Kamgar, et al. "Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma." Cancer Research, vol. 86, no. 8, 2026, p. 2042, https://doi.org/10.1158/0008-5472.CAN-25-0018.