Mycobacterium tuberculosis triggers reduced inflammatory cytokine responses and virulence in mice lacking Tax1bp1.

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Title: Mycobacterium tuberculosis triggers reduced inflammatory cytokine responses and virulence in mice lacking Tax1bp1.
Authors: Chin J; Department of Medicine, University of California, San Francisco, California, United States of America., Abeydeera N; Department of Medicine, University of California, San Francisco, California, United States of America., Repasy T; Department of Molecular and Cell Biology, University of California, Berkeley, California, United States of America., Rivera-Lugo R; Department of Molecular and Cell Biology, University of California, Berkeley, California, United States of America., Mitchell G; Department of Molecular and Cell Biology, University of California, Berkeley, California, United States of America., Nguyen VQ; Gladstone-UCSF Institute of Genomic Immunology, San Francisco, California, United States of America.; Department of Surgery, University of California, San Francisco, California, United States of America.; Diabetes Center, University of California, San Francisco, California, United States of America.; UCSF CoLabs, University of California, San Francisco, California, United States of America., Zheng W; Division of Experimental Medicine, Department of Medicine, University of California, San Francisco, California, United States of America., Richards A; Quantitative Biosciences Institute (QBI), University of California, San Francisco, San Francisco, California, United States of America.; J. David Gladstone Institutes, San Francisco, California, United States of America.; Department of Bioengineering and Therapeutic Sciences, University of California, San Francisco, California, United States of America., Stevenson E; Quantitative Biosciences Institute (QBI), University of California, San Francisco, San Francisco, California, United States of America.; J. David Gladstone Institutes, San Francisco, California, United States of America.; Department of Bioengineering and Therapeutic Sciences, University of California, San Francisco, California, United States of America., Swaney DL; Quantitative Biosciences Institute (QBI), University of California, San Francisco, San Francisco, California, United States of America.; J. David Gladstone Institutes, San Francisco, California, United States of America.; Department of Bioengineering and Therapeutic Sciences, University of California, San Francisco, California, United States of America., Krogan NJ; Quantitative Biosciences Institute (QBI), University of California, San Francisco, San Francisco, California, United States of America.; J. David Gladstone Institutes, San Francisco, California, United States of America.; Department of Bioengineering and Therapeutic Sciences, University of California, San Francisco, California, United States of America., Ernst JD; Division of Experimental Medicine, Department of Medicine, University of California, San Francisco, California, United States of America., Cox JS; Department of Molecular and Cell Biology, University of California, Berkeley, California, United States of America., Budzik JM; Department of Medicine, University of California, San Francisco, California, United States of America.; Department of Molecular and Cell Biology, University of California, Berkeley, California, United States of America.
Source: PLoS pathogens [PLoS Pathog] 2025 Oct 31; Vol. 21 (10), pp. e1012829. Date of Electronic Publication: 2025 Oct 31 (Print Publication: 2025).
Publication Type: Journal Article
Journal Info: Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet ISSN: 1553-7374 (Electronic) Linking ISSN: 15537366 NLM ISO Abbreviation: PLoS Pathog Subsets: MEDLINE
Database: MEDLINE Ultimate
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ISSN:1553-7374
DOI:10.1371/journal.ppat.1012829