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Syntaphilin loss enhances mitochondrial axonal transport and neuromuscular junction formation in a human stem cell derived neuromuscular assembloid model.

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Title: Syntaphilin loss enhances mitochondrial axonal transport and neuromuscular junction formation in a human stem cell derived neuromuscular assembloid model.
Authors: Salzinger A; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK., Özkan E; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK., Ramesh V; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK., Nanda J; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK., Burr K; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK., Story D; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK., Pham NT; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Institute for Regeneration and Repair, University of Edinburgh, Edinburgh, UK., Chandran S; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK., Selvaraj BT; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK. bthangar@exseed.ed.ac.uk.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK. bthangar@exseed.ed.ac.uk.
Source: Molecular medicine (Cambridge, Mass.) [Mol Med] 2025 Nov 05; Vol. 31 (1), pp. 328. Date of Electronic Publication: 2025 Nov 05.
Publication Type: Journal Article
Journal Info: Publisher: BioMed Central Country of Publication: England NLM ID: 9501023 Publication Model: Electronic Cited Medium: Internet ISSN: 1528-3658 (Electronic) Linking ISSN: 10761551 NLM ISO Abbreviation: Mol Med Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Syntaphilin loss enhances mitochondrial axonal transport and neuromuscular junction formation in a human stem cell derived neuromuscular assembloid model.
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  Data: <searchLink fieldCode="AU" term="%22Salzinger+A%22">Salzinger A</searchLink>; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK.<br /><searchLink fieldCode="AU" term="%22Özkan+E%22">Özkan E</searchLink>; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK.<br /><searchLink fieldCode="AU" term="%22Ramesh+V%22">Ramesh V</searchLink>; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK.<br /><searchLink fieldCode="AU" term="%22Nanda+J%22">Nanda J</searchLink>; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK.<br /><searchLink fieldCode="AU" term="%22Burr+K%22">Burr K</searchLink>; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK.<br /><searchLink fieldCode="AU" term="%22Story+D%22">Story D</searchLink>; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK.<br /><searchLink fieldCode="AU" term="%22Pham+NT%22">Pham NT</searchLink>; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Institute for Regeneration and Repair, University of Edinburgh, Edinburgh, UK.<br /><searchLink fieldCode="AU" term="%22Chandran+S%22">Chandran S</searchLink>; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK.<br /><searchLink fieldCode="AU" term="%22Selvaraj+BT%22">Selvaraj BT</searchLink>; UK Dementia Research Institute at University of Edinburgh , Edinburgh, UK. bthangar@exseed.ed.ac.uk.; Centre for Clinical Brain Sciences and Euan McDonald Centre for MND Research, University of Edinburgh, Edinburgh, UK. bthangar@exseed.ed.ac.uk.
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  Data: <searchLink fieldCode="JN" term="%229501023%22">Molecular medicine (Cambridge, Mass.)</searchLink> [Mol Med] 2025 Nov 05; Vol. 31 (1), pp. 328. <i>Date of Electronic Publication: </i>2025 Nov 05.
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              Text: 2025 Nov 05
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