HIV-induced sialoglycans on infected CD4+ T cells promote immune evasion from myeloid cell-mediated killing.
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| Title: | HIV-induced sialoglycans on infected CD4+ T cells promote immune evasion from myeloid cell-mediated killing. |
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| Authors: | Singh S; Division of Infectious Diseases, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA.; Center for Human Immunobiology, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA.; Potocsnak Longevity Institute, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA., Islam SMS; Division of Infectious Diseases, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA.; Center for Human Immunobiology, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA.; Potocsnak Longevity Institute, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA., Liu R; Rice University, Houston, TX, USA., Adeniji OS; Fred Hutchinson Cancer Center, Seattle, WA, USA., Simons LM; Division of Infectious Diseases, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA., Saini P; Division of Infectious Diseases, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA.; Center for Human Immunobiology, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA.; Potocsnak Longevity Institute, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA., Tateno H; Cellular and Molecular Biotechnology Research Institute, National Institute of Advanced Industrial Science and Technology (AIST), Tsukuba, Ibaraki, Japan., Danesh A; Weill Cornell Medicine, Department of Medicine, Division of Infectious Diseases, New York, NY, USA., Denton PW; University of Nebraska Omaha, Omaha, NE, USA., Giron LB; Division of Infectious Diseases, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA.; Center for Human Immunobiology, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA.; Potocsnak Longevity Institute, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA., Jones RB; Weill Cornell Medicine, Department of Medicine, Division of Infectious Diseases, New York, NY, USA., Hultquist JF; Division of Infectious Diseases, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA., Xiao H; Rice University, Houston, TX, USA., Abdel-Mohsen M; Division of Infectious Diseases, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA. mmohsen@northwestern.edu.; Center for Human Immunobiology, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA. mmohsen@northwestern.edu.; Potocsnak Longevity Institute, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA. mmohsen@northwestern.edu. |
| Source: | Nature communications [Nat Commun] 2025 Dec 13; Vol. 17 (1), pp. 18. Date of Electronic Publication: 2025 Dec 13. |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101528555 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-1723 (Electronic) Linking ISSN: 20411723 NLM ISO Abbreviation: Nat Commun Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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