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The renal response to FGF23 shifts from phosphaturia toward inflammation in kidney disease.

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Title: The renal response to FGF23 shifts from phosphaturia toward inflammation in kidney disease.
Authors: Moor MB; Division of Renal Medicine Department of Clinical Science Intervention and Technology Karolinska Institutet Stockholm Sweden.; Division of Pathology Department of Laboratory Medicine Karolinska Institutet Stockholm Sweden.; Department of Nephrology and Hypertension University Hospital Bern and University of Bern Bern Switzerland., Burmakin M; Division of Pathology Department of Laboratory Medicine Karolinska Institutet Stockholm Sweden., Levin A; Division of Renal Medicine Department of Clinical Science Intervention and Technology Karolinska Institutet Stockholm Sweden., Korkut GG; Division of Pathology Department of Laboratory Medicine Karolinska Institutet Stockholm Sweden., Brodin D; Bioinformatics and Expression Analysis Core Facility (BEA) Karolinska Institutet Huddinge Sweden., Wernerson A; Division of Renal Medicine Department of Clinical Science Intervention and Technology Karolinska Institutet Stockholm Sweden., Bruchfeld A; Division of Renal Medicine Department of Clinical Science Intervention and Technology Karolinska Institutet Stockholm Sweden.; Department of Health Medicine and Caring Sciences Linköping University Linköping Sweden., Bárány P; Division of Renal Medicine Department of Clinical Science Intervention and Technology Karolinska Institutet Stockholm Sweden., Witasp A; Division of Renal Medicine Department of Clinical Science Intervention and Technology Karolinska Institutet Stockholm Sweden., Patrakka J; Division of Pathology Department of Laboratory Medicine Karolinska Institutet Stockholm Sweden., Olauson H; Division of Pathology Department of Laboratory Medicine Karolinska Institutet Stockholm Sweden.
Source: Journal of cell communication and signaling [J Cell Commun Signal] 2026 Feb 23; Vol. 20 (1), pp. e70061. Date of Electronic Publication: 2026 Feb 23 (Print Publication: 2026).
Publication Type: Journal Article
Journal Info: Publisher: Wiley-Blackwell Country of Publication: United States NLM ID: 101308338 Publication Model: eCollection Cited Medium: Print ISSN: 1873-9601 (Print) Linking ISSN: 18739601 NLM ISO Abbreviation: J Cell Commun Signal Subsets: PubMed not MEDLINE
Database: MEDLINE Ultimate
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  Data: The renal response to FGF23 shifts from phosphaturia toward inflammation in kidney disease.
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  Data: <searchLink fieldCode="AU" term="%22Moor+MB%22">Moor MB</searchLink>; Division of Renal Medicine Department of Clinical Science Intervention and Technology Karolinska Institutet Stockholm Sweden.; Division of Pathology Department of Laboratory Medicine Karolinska Institutet Stockholm Sweden.; Department of Nephrology and Hypertension University Hospital Bern and University of Bern Bern Switzerland.<br /><searchLink fieldCode="AU" term="%22Burmakin+M%22">Burmakin M</searchLink>; Division of Pathology Department of Laboratory Medicine Karolinska Institutet Stockholm Sweden.<br /><searchLink fieldCode="AU" term="%22Levin+A%22">Levin A</searchLink>; Division of Renal Medicine Department of Clinical Science Intervention and Technology Karolinska Institutet Stockholm Sweden.<br /><searchLink fieldCode="AU" term="%22Korkut+GG%22">Korkut GG</searchLink>; Division of Pathology Department of Laboratory Medicine Karolinska Institutet Stockholm Sweden.<br /><searchLink fieldCode="AU" term="%22Brodin+D%22">Brodin D</searchLink>; Bioinformatics and Expression Analysis Core Facility (BEA) Karolinska Institutet Huddinge Sweden.<br /><searchLink fieldCode="AU" term="%22Wernerson+A%22">Wernerson A</searchLink>; Division of Renal Medicine Department of Clinical Science Intervention and Technology Karolinska Institutet Stockholm Sweden.<br /><searchLink fieldCode="AU" term="%22Bruchfeld+A%22">Bruchfeld A</searchLink>; Division of Renal Medicine Department of Clinical Science Intervention and Technology Karolinska Institutet Stockholm Sweden.; Department of Health Medicine and Caring Sciences Linköping University Linköping Sweden.<br /><searchLink fieldCode="AU" term="%22Bárány+P%22">Bárány P</searchLink>; Division of Renal Medicine Department of Clinical Science Intervention and Technology Karolinska Institutet Stockholm Sweden.<br /><searchLink fieldCode="AU" term="%22Witasp+A%22">Witasp A</searchLink>; Division of Renal Medicine Department of Clinical Science Intervention and Technology Karolinska Institutet Stockholm Sweden.<br /><searchLink fieldCode="AU" term="%22Patrakka+J%22">Patrakka J</searchLink>; Division of Pathology Department of Laboratory Medicine Karolinska Institutet Stockholm Sweden.<br /><searchLink fieldCode="AU" term="%22Olauson+H%22">Olauson H</searchLink>; Division of Pathology Department of Laboratory Medicine Karolinska Institutet Stockholm Sweden.
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  Data: <searchLink fieldCode="JN" term="%22101308338%22">Journal of cell communication and signaling</searchLink> [J Cell Commun Signal] 2026 Feb 23; Vol. 20 (1), pp. e70061. <i>Date of Electronic Publication: </i>2026 Feb 23 (<i>Print Publication: </i>2026).
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Wiley-Blackwell%22">Wiley-Blackwell </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>101308338 <i>Publication Model: </i>eCollection <i>Cited Medium: </i>Print <i>ISSN: </i>1873-9601 (Print) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2218739601%22">18739601 </searchLink><i>NLM ISO Abbreviation: </i>J Cell Commun Signal <i>Subsets: </i>PubMed not MEDLINE
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=41744000
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              Text: 2026 Feb 23
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