BACH2 ameliorates cell apoptosis and autophagy as a molecular switch of BCL-2/Beclin-1 complex in myocardial infarction.

Saved in:
Bibliographic Details
Title: BACH2 ameliorates cell apoptosis and autophagy as a molecular switch of BCL-2/Beclin-1 complex in myocardial infarction.
Authors: Wang X; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Zhang H; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Liu X; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., He X; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Cao M; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Wang W; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Yin C; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Li L; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Wang S; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Yang C; Department of Cardiac Surgery, Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, 200032, China. yang.cheng@zs-hospital.sh.cn., Gao P; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China. gaop11@fudan.edu.cn., Zou Y; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China. zou.yunzeng@zs-hospital.sh.cn.; Institutes of Advanced Medical Sciences and Huaihe Hospital, Henan University, Kaifeng, 475000, Henan, China. zou.yunzeng@zs-hospital.sh.cn.; Qing Pu Branch of Zhongshan Hospital, Fudan University, Shanghai, China. zou.yunzeng@zs-hospital.sh.cn.
Source: Apoptosis : an international journal on programmed cell death [Apoptosis] 2026 Mar 15; Vol. 31 (3). Date of Electronic Publication: 2026 Mar 15.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Springer Country of Publication: Netherlands NLM ID: 9712129 Publication Model: Electronic Cited Medium: Internet ISSN: 1573-675X (Electronic) Linking ISSN: 13608185 NLM ISO Abbreviation: Apoptosis Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1573-675X
DOI:10.1007/s10495-026-02317-y