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BACH2 ameliorates cell apoptosis and autophagy as a molecular switch of BCL-2/Beclin-1 complex in myocardial infarction.

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Title: BACH2 ameliorates cell apoptosis and autophagy as a molecular switch of BCL-2/Beclin-1 complex in myocardial infarction.
Authors: Wang X; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Zhang H; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Liu X; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., He X; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Cao M; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Wang W; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Yin C; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Li L; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Wang S; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China., Yang C; Department of Cardiac Surgery, Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, 200032, China. yang.cheng@zs-hospital.sh.cn., Gao P; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China. gaop11@fudan.edu.cn., Zou Y; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China. zou.yunzeng@zs-hospital.sh.cn.; Institutes of Advanced Medical Sciences and Huaihe Hospital, Henan University, Kaifeng, 475000, Henan, China. zou.yunzeng@zs-hospital.sh.cn.; Qing Pu Branch of Zhongshan Hospital, Fudan University, Shanghai, China. zou.yunzeng@zs-hospital.sh.cn.
Source: Apoptosis : an international journal on programmed cell death [Apoptosis] 2026 Mar 15; Vol. 31 (3). Date of Electronic Publication: 2026 Mar 15.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Springer Country of Publication: Netherlands NLM ID: 9712129 Publication Model: Electronic Cited Medium: Internet ISSN: 1573-675X (Electronic) Linking ISSN: 13608185 NLM ISO Abbreviation: Apoptosis Subsets: MEDLINE
Database: MEDLINE Ultimate
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PubType: Academic Journal
PubTypeId: academicJournal
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  Data: BACH2 ameliorates cell apoptosis and autophagy as a molecular switch of BCL-2/Beclin-1 complex in myocardial infarction.
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  Data: <searchLink fieldCode="AU" term="%22Wang+X%22">Wang X</searchLink>; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China.<br /><searchLink fieldCode="AU" term="%22Zhang+H%22">Zhang H</searchLink>; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China.<br /><searchLink fieldCode="AU" term="%22Liu+X%22">Liu X</searchLink>; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China.<br /><searchLink fieldCode="AU" term="%22He+X%22">He X</searchLink>; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China.<br /><searchLink fieldCode="AU" term="%22Cao+M%22">Cao M</searchLink>; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China.<br /><searchLink fieldCode="AU" term="%22Wang+W%22">Wang W</searchLink>; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China.<br /><searchLink fieldCode="AU" term="%22Yin+C%22">Yin C</searchLink>; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China.<br /><searchLink fieldCode="AU" term="%22Li+L%22">Li L</searchLink>; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China.<br /><searchLink fieldCode="AU" term="%22Wang+S%22">Wang S</searchLink>; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China.<br /><searchLink fieldCode="AU" term="%22Yang+C%22">Yang C</searchLink>; Department of Cardiac Surgery, Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, 200032, China. yang.cheng@zs-hospital.sh.cn.<br /><searchLink fieldCode="AU" term="%22Gao+P%22">Gao P</searchLink>; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China. gaop11@fudan.edu.cn.<br /><searchLink fieldCode="AU" term="%22Zou+Y%22">Zou Y</searchLink>; Shanghai Institute of Cardiovascular Diseases and State Key Laboratory of Cardiovascular Diseases, Zhongshan Hospital and Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, China. zou.yunzeng@zs-hospital.sh.cn.; Institutes of Advanced Medical Sciences and Huaihe Hospital, Henan University, Kaifeng, 475000, Henan, China. zou.yunzeng@zs-hospital.sh.cn.; Qing Pu Branch of Zhongshan Hospital, Fudan University, Shanghai, China. zou.yunzeng@zs-hospital.sh.cn.
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  Data: <searchLink fieldCode="JN" term="%229712129%22">Apoptosis : an international journal on programmed cell death</searchLink> [Apoptosis] 2026 Mar 15; Vol. 31 (3). <i>Date of Electronic Publication: </i>2026 Mar 15.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Springer%22">Springer </searchLink><i>Country of Publication: </i>Netherlands <i>NLM ID: </i>9712129 <i>Publication Model: </i>Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1573-675X (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2213608185%22">13608185 </searchLink><i>NLM ISO Abbreviation: </i>Apoptosis <i>Subsets: </i>MEDLINE
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        Value: 10.1007/s10495-026-02317-y
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      – TitleFull: BACH2 ameliorates cell apoptosis and autophagy as a molecular switch of BCL-2/Beclin-1 complex in myocardial infarction.
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            – D: 15
              M: 03
              Text: 2026 Mar 15
              Type: published
              Y: 2026
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            – TitleFull: Apoptosis : an international journal on programmed cell death
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